Assessment of Vascular Dysfunction and Inflammation Induced by Angiotensin II in Mice.

about

Assessment of Vascular Dysfunction and Inflammation Induced by Angiotensin II in Mice.

Item

http://www.wikidata.org/entity/Q52930674

description

2017 nî lūn-bûn

@nan

2017年の論文

@ja

2017年学术文章

@wuu

2017年学术文章

@zh

2017年学术文章

@zh-cn

2017年学术文章

@zh-hans

2017年学术文章

@zh-my

2017年学术文章

@zh-sg

2017年學術文章

@yue

2017年學術文章

@zh-hant

name

Assessment of Vascular Dysfunction and Inflammation Induced by Angiotensin II in Mice.

@en

Assessment of Vascular Dysfunction and Inflammation Induced by Angiotensin II in Mice.

@nl

type

Item

label

Assessment of Vascular Dysfunction and Inflammation Induced by Angiotensin II in Mice.

@en

Assessment of Vascular Dysfunction and Inflammation Induced by Angiotensin II in Mice.

@nl

prefLabel

Assessment of Vascular Dysfunction and Inflammation Induced by Angiotensin II in Mice.

@en

Assessment of Vascular Dysfunction and Inflammation Induced by Angiotensin II in Mice.

@nl

P1476

Assessment of Vascular Dysfunction and Inflammation Induced by Angiotensin II in Mice.

@en

P2093

Jeremy Lagrange

http://www.w3.org/2001/XMLSchema#string

Philip Wenzel

http://www.w3.org/2001/XMLSchema#string

P2860

Identification of splenic reservoir monocytes and their deployment to inflammatory sites

Angiotensin II-induced hypertension accelerates the development of atherosclerosis in apoE-deficient mice.

Inflammatory monocytes determine endothelial nitric-oxide synthase uncoupling and nitro-oxidative stress induced by angiotensin II

Vascular inflammatory cells in hypertension.

Role of the T cell in the genesis of angiotensin II induced hypertension and vascular dysfunction.

NADPH oxidases in cardiovascular health and disease.

Heme oxygenase-1 suppresses a pro-inflammatory phenotype in monocytes and determines endothelial function and arterial hypertension in mice and humans.

Distinct infiltration of neutrophils in lesion shoulders in ApoE-/- mice.

Angiotensin II-induced vascular dysfunction depends on interferon-γ-driven immune cell recruitment and mutual activation of monocytes and NK-cells.

ApoE(-/-)/lysozyme M(EGFP/EGFP) mice as a versatile model to study monocyte and neutrophil trafficking in atherosclerosis.

P304

439-453

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

P356

10.1007/978-1-4939-6786-5_30

http://www.w3.org/2001/XMLSchema#string

P407

English

P478

1559

http://www.w3.org/2001/XMLSchema#string

P50

Sabine Kossmann

P577

2017-01-01T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P698

28063062

http://www.w3.org/2001/XMLSchema#string