Combined deletion of Vhl, Trp53 and Kif3a causes cystic and neoplastic renal lesions.

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Role of primary cilia in non-dividing and post-mitotic cells.Combined mutation in Vhl, Trp53 and Rb1 causes clear cell renal cell carcinoma in mice.Generation of autochthonous mouse models of clear cell renal cell carcinoma: mouse models of renal cell carcinoma.

P2860

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P2860

Combined deletion of Vhl, Trp53 and Kif3a causes cystic and neoplastic renal lesions.

Item

http://www.wikidata.org/entity/Q53112209

description

2016 nî lūn-bûn

@nan

2016年の論文

@ja

2016年学术文章

@wuu

2016年学术文章

@zh

2016年学术文章

@zh-cn

2016年学术文章

@zh-hans

2016年学术文章

@zh-my

2016年学术文章

@zh-sg

2016年學術文章

@yue

2016年學術文章

@zh-hant

name

Combined deletion of Vhl, Trp53 and Kif3a causes cystic and neoplastic renal lesions.

@en

Combined deletion of Vhl, Trp53 and Kif3a causes cystic and neoplastic renal lesions.

@nl

type

Item

label

Combined deletion of Vhl, Trp53 and Kif3a causes cystic and neoplastic renal lesions.

@en

Combined deletion of Vhl, Trp53 and Kif3a causes cystic and neoplastic renal lesions.

@nl

prefLabel

Combined deletion of Vhl, Trp53 and Kif3a causes cystic and neoplastic renal lesions.

@en

Combined deletion of Vhl, Trp53 and Kif3a causes cystic and neoplastic renal lesions.

@nl

P1476

Combined deletion of Vhl, Trp53 and Kif3a causes cystic and neoplastic renal lesions.

@en

P2093

Anna Guinot

http://www.w3.org/2001/XMLSchema#string

Holger Lehmann

http://www.w3.org/2001/XMLSchema#string

Peter J Wild

http://www.w3.org/2001/XMLSchema#string

P2860

Comprehensive molecular characterization of clear cell renal cell carcinoma.

Integrated molecular analysis of clear-cell renal cell carcinoma.

Molecular basis of the VHL hereditary cancer syndrome

The primary cilium as a complex signaling center

VHL-gene deletion in single renal tubular epithelial cells and renal tubular cysts: further evidence for a cyst-dependent progression pathway of clear cell renal carcinoma in von Hippel-Lindau disease.

An incredible decade for the primary cilium: a look at a once-forgotten organelle.

Bap1 is essential for kidney function and cooperates with Vhl in renal tumorigenesis

Loss of primary cilia upregulates renal hypertrophic signaling and promotes cystogenesis.

Kidney-specific inactivation of the KIF3A subunit of kinesin-II inhibits renal ciliogenesis and produces polycystic kidney disease

Acute kidney injury and aberrant planar cell polarity induce cyst formation in mice lacking renal cilia.

P304

365-373

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scholarly article

P356

10.1002/PATH.4736

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P478

239

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P50

Ian J Frew

P577

2016-05-30T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P698

27126173

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Combined deletion of Vhl, Trp53 and Kif3a causes cystic and neoplastic renal lesions.

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P2860

P2860

Combined deletion of Vhl, Trp53 and Kif3a causes cystic and neoplastic renal lesions.

description

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prefLabel

P1433

P1476

P2093

P2860

P304

P31

P356

P433

P478

P50

P577

P698

P356

P1433

P1476

P2093

P2860

P304

P31

P356

P433

P478

P50

P577

P698