Renovascular remodeling and renal injury after extended angiotensin II infusion.
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ENaC inhibition stimulates HCl secretion in the mouse cortical collecting duct. II. Bafilomycin-sensitive H+ secretionEndothelial Epas1 Deficiency Is Sufficient To Promote Parietal Epithelial Cell Activation and FSGS in Experimental Hypertension.Endothelial mineralocorticoid receptor ablation does not alter blood pressure, kidney function or renal vessel contractility.Dysregulation of the Renin-Angiotensin System and the Vasopressinergic System Interactions in Cardiovascular Disorders.Angiotensin II-induced podocyte apoptosis is mediated by endoplasmic reticulum stress/PKC-δ/p38 MAPK pathway activation and trough increased Na+/H+ exchanger isoform 1 activity.
P2860
Renovascular remodeling and renal injury after extended angiotensin II infusion.
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2016 nî lūn-bûn
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2016年の論文
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2016年学术文章
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2016年学术文章
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2016年学术文章
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2016年学术文章
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2016年学术文章
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2016年学术文章
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2016年學術文章
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2016年學術文章
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name
Renovascular remodeling and renal injury after extended angiotensin II infusion.
@en
Renovascular remodeling and renal injury after extended angiotensin II infusion.
@nl
type
label
Renovascular remodeling and renal injury after extended angiotensin II infusion.
@en
Renovascular remodeling and renal injury after extended angiotensin II infusion.
@nl
prefLabel
Renovascular remodeling and renal injury after extended angiotensin II infusion.
@en
Renovascular remodeling and renal injury after extended angiotensin II infusion.
@nl
P2093
P2860
P1476
Renovascular remodeling and renal injury after extended angiotensin II infusion.
@en
P2093
Fernando Augusto Malavazzi Casare
Gisele Kruger Couto
Juliana Martins Costa-Pessoa
Karina Thieme
Luciana Venturini Rossoni
Maria Oliveira-Souza
P2860
P304
P356
10.1152/AJPRENAL.00471.2015
P577
2016-03-09T00:00:00Z