High glucose-induced hypertrophy of mesangial cells is reversed by connexin43 overexpression via PTEN/Akt/mTOR signaling.
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Pericytes, an overlooked player in vascular pathobiologyAdvanced Glycation End Product (AGE)-AGE Receptor (RAGE) System Upregulated Connexin43 Expression in Rat Cardiomyocytes via PKC and Erk MAPK PathwaysDown-regulation of miR-34a alleviates mesangial proliferation in vitro and glomerular hypertrophy in early diabetic nephropathy mice by targeting GAS1Never in mitosis gene A related kinase-6 attenuates pressure overload-induced activation of the protein kinase B pathway and cardiac hypertrophyMetallothionein-I/II Knockout Mice Aggravate Mitochondrial Superoxide Production and Peroxiredoxin 3 Expression in Thyroid after Excessive Iodide Exposure.Kidney glycosphingolipids are elevated early in diabetic nephropathy and mediate hypertrophy of mesangial cellsHydrophobic motif site-phosphorylated protein kinase CβII between mTORC2 and Akt regulates high glucose-induced mesangial cell hypertrophy.Connexin43 mediates NF-κB signalling activation induced by high glucose in GMCs: involvement of c-Src.Cross talk between miR-214 and PTEN attenuates glomerular hypertrophy under diabetic conditions.Astragaloside IV ameliorates renal injury in db/db mice.Mind the gap: connexins and cell-cell communication in the diabetic kidney.Pleiotrophin, a target of miR-384, promotes proliferation, metastasis and lipogenesis in HBV-related hepatocellular carcinoma.Effect of Novel Gasotransmitter hydrogen sulfide on renal fibrosis and connexins expression in diabetic ratsp21(WAF1/CIP1) Expression is Differentially Regulated by Metformin and Rapamycin.Alpha Lipoic Acid Modulated High Glucose-Induced Rat Mesangial Cell Dysfunction via mTOR/p70S6K/4E-BP1 Pathway.High glucose induces the aging of mesenchymal stem cells via Akt/mTOR signaling.Consequences of calorie restriction and calorie excess for the physiological parameters of the yeast Saccharomyces cerevisiae cells.Angiotensin II-Induced Mesangial Cell Damaged Is Preceded by Cell Membrane Permeabilization Due to Upregulation of Non-Selective Channels.Rosiglitazone diminishes the high-glucose-induced modulation of 5-fluorouracil cytotoxicity in colorectal cancer cells.
P2860
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P2860
High glucose-induced hypertrophy of mesangial cells is reversed by connexin43 overexpression via PTEN/Akt/mTOR signaling.
description
2011 nî lūn-bûn
@nan
2011年の論文
@ja
2011年学术文章
@wuu
2011年学术文章
@zh
2011年学术文章
@zh-cn
2011年学术文章
@zh-hans
2011年学术文章
@zh-my
2011年学术文章
@zh-sg
2011年學術文章
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2011年學術文章
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name
High glucose-induced hypertrop ...... n via PTEN/Akt/mTOR signaling.
@en
High glucose-induced hypertrop ...... n via PTEN/Akt/mTOR signaling.
@nl
type
label
High glucose-induced hypertrop ...... n via PTEN/Akt/mTOR signaling.
@en
High glucose-induced hypertrop ...... n via PTEN/Akt/mTOR signaling.
@nl
prefLabel
High glucose-induced hypertrop ...... n via PTEN/Akt/mTOR signaling.
@en
High glucose-induced hypertrop ...... n via PTEN/Akt/mTOR signaling.
@nl
P2093
P2860
P356
P1476
High glucose-induced hypertrop ...... n via PTEN/Akt/mTOR signaling.
@en
P2093
Guang-Yan Cai
Jian-Jun Gao
Shao-Yuan Cui
Xiang-Mei Chen
P2860
P304
P356
10.1093/NDT/GFR265
P407
P577
2011-06-01T00:00:00Z