Loss of gene function as a consequence of human papillomavirus DNA integration. - Wikidata - awgldk - TriplyDB

Loss of gene function as a consequence of human papillomavirus DNA integration.

Loss of gene function as a consequence of human papillomavirus DNA integration.

http://www.wikidata.org/entity/Q53275559

about

Identification of episomal human papillomavirus and other DNA viruses in cytological anal samples of HIV-uninfected men who have sex with men.Reduction in the copy number and expression level of the recurrent human papillomavirus integration gene fragile histidine triad (FHIT) predicts the transition of cervical lesions.Mobile elements and viral integrations prompt considerations for bacterial DNA integration as a novel carcinogen Non-random integration of the HPV genome in cervical cancer.Characterization of HPV and host genome interactions in primary head and neck cancers.Multiplex Identification of Human Papillomavirus 16 DNA Integration Sites in Cervical Carcinomas.Comprehensive analysis of HPV16 integration in OSCC reveals no significant impact of physical status on viral oncogene and virally disrupted human gene expression.Transcriptionally active regions are the preferred targets for chromosomal HPV integration in cervical carcinogenesis.Chromosomal copy number alterations and HPV integration in cervical precancer and invasive cancer Evidence of disrupted high-risk human papillomavirus DNA in morphologically normal cervices of older women.CASZ1 inhibits cell cycle progression in neuroblastoma by restoring pRb activity Dysregulation of host cellular genes targeted by human papillomavirus (HPV) integration contributes to HPV-related cervical carcinogenesis.HPV16 integration probably contributes to cervical oncogenesis through interrupting tumor suppressor genes and inducing chromosome instability.Identification of CASZ1 NES reveals potential mechanisms for loss of CASZ1 tumor suppressor activity in neuroblastoma.Genome-wide analysis of HPV integration in human cancers reveals recurrent, focal genomic instability RUNX3 and CAMK2N1 hypermethylation as prognostic marker for epithelial ovarian cancer.Genomic characterization of viral integration sites in HPV-related cancers.Genome-wide profiling of HPV integration in cervical cancer identifies clustered genomic hot spots and a potential microhomology-mediated integration mechanism.Induction of dormancy in hypoxic human papillomavirus-positive cancer cells.Viral-Cellular DNA Junctions as Molecular Markers for Assessing Intra-Tumor Heterogeneity in Cervical Cancer and for the Detection of Circulating Tumor DNA.Development of safer gene delivery systems to minimize the risk of insertional mutagenesis-related malignancies: a critical issue for the field of gene therapy.The precision prevention and therapy of HPV-related cervical cancer: new concepts and clinical implications

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Loss of gene function as a consequence of human papillomavirus DNA integration.

http://www.wikidata.org/entity/Q53275559

description

2012 nî lūn-bûn

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2012年の論文

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2012年学术文章

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2012年学术文章

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2012年学术文章

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2012年学术文章

@zh-hans

2012年学术文章

@zh-my

2012年学术文章

@zh-sg

2012年學術文章

@yue

2012年學術文章

@zh-hant

name

Loss of gene function as a consequence of human papillomavirus DNA integration.

@en

Loss of gene function as a consequence of human papillomavirus DNA integration.

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type

label

Loss of gene function as a consequence of human papillomavirus DNA integration.

@en

Loss of gene function as a consequence of human papillomavirus DNA integration.

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prefLabel

Loss of gene function as a consequence of human papillomavirus DNA integration.

@en

Loss of gene function as a consequence of human papillomavirus DNA integration.

@nl

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International Journal of Cancer

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Loss of gene function as a consequence of human papillomavirus DNA integration.

@en

P2093

Corina Driesch

http://www.w3.org/2001/XMLSchema#string

Ingo B Runnebaum

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Katrin Beer-Grondke

http://www.w3.org/2001/XMLSchema#string

Lars Jansen

http://www.w3.org/2001/XMLSchema#string

Martina Schmitz

http://www.w3.org/2001/XMLSchema#string

Matthias Dürst

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P2860

Papillomaviruses causing cancer: evasion from host-cell control in early events in carcinogenesis

Common fragile sites are preferential targets for HPV16 integrations in cervical tumors

Epidemiologic classification of human papillomavirus types associated with cervical cancer

APM-1, a novel human gene, identified by aberrant co-transcription with papillomavirus oncogenes in a cervical carcinoma cell line, encodes a BTB/POZ-zinc finger protein with growth inhibitory activity.

Mechanism of genomic instability in cells infected with the high-risk human papillomaviruses.

CASZ1b, the short isoform of CASZ1 gene, coexpresses with CASZ1a during neurogenesis and suppresses neuroblastoma cell growth

Integrated human papillomavirus type 16 is frequently found in cervical cancer precursors as demonstrated by a novel quantitative real-time PCR technique.

Human papillomavirus immortalization and transformation functions.

The human papillomavirus E6 protein and its contribution to malignant progression.

Molecular cloning and characterization of human Castor, a novel human gene upregulated during cell differentiation.

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E593-602

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scholarly article

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10.1002/IJC.27433

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5

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131

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2012-02-22T00:00:00Z

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221763908

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22262398

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