Inverse genetic predisposition to colon versus lung carcinogenesis in mouse lines selected based on acute inflammatory responsiveness.
about
Gene expression profiles of bone marrow cells from mice phenotype-selected for maximal or minimal acute inflammations: searching for genes in acute inflammation modifier loci.Genetic control of renal tumorigenesis by the mouse Rtm1 locus.Trypanosoma cruzi infection in genetically selected mouse lines: genetic linkage with quantitative trait locus controlling antibody response.Pristane-induced arthritis loci interact with the Slc11a1 gene to determine susceptibility in mice selected for high inflammationPolymorphonuclear neutrophils and cancer: intense and sustained neutrophilia as a treatment against solid tumors.Slc11a1 (Nramp-1) gene modulates immune-inflammation genes in macrophages during pristane-induced arthritis in mice.Acute Inflammation Loci Are Involved in Wound Healing in the Mouse Ear Punch Model.
P2860
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P2860
Inverse genetic predisposition to colon versus lung carcinogenesis in mouse lines selected based on acute inflammatory responsiveness.
description
2006 nî lūn-bûn
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2006年の論文
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2006年学术文章
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name
Inverse genetic predisposition ...... e inflammatory responsiveness.
@en
Inverse genetic predisposition ...... e inflammatory responsiveness.
@nl
type
label
Inverse genetic predisposition ...... e inflammatory responsiveness.
@en
Inverse genetic predisposition ...... e inflammatory responsiveness.
@nl
prefLabel
Inverse genetic predisposition ...... e inflammatory responsiveness.
@en
Inverse genetic predisposition ...... e inflammatory responsiveness.
@nl
P2093
P50
P356
P1433
P1476
Inverse genetic predisposition ...... te inflammatory responsiveness
@en
P2093
Michel Seman
Orlando Garcia Ribeiro
Roberto Francisco Di Pace
Solange Massa
Wafa Hanna Koury Cabrera
P304
P356
10.1093/CARCIN/BGL080
P407
P577
2006-06-13T00:00:00Z