Toll-like receptor 5 deficiency exacerbates cardiac injury and inflammation induced by myocardial ischaemia-reperfusion in the mouse.

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Leukocyte TLR5 deficiency inhibits atherosclerosis by reduced macrophage recruitment and defective T-cell responsiveness.The Biological Basis for Cardiac Repair After Myocardial Infarction: From Inflammation to Fibrosis.Insights into innate immune signalling in controlling cardiac remodelling.

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Toll-like receptor 5 deficiency exacerbates cardiac injury and inflammation induced by myocardial ischaemia-reperfusion in the mouse.

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2015年の論文

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2015年学术文章

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Toll-like receptor 5 deficienc ...... emia-reperfusion in the mouse.

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Toll-like receptor 5 deficienc ...... emia-reperfusion in the mouse.

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Item

label

Toll-like receptor 5 deficienc ...... emia-reperfusion in the mouse.

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Toll-like receptor 5 deficienc ...... emia-reperfusion in the mouse.

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Toll-like receptor 5 deficienc ...... emia-reperfusion in the mouse.

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Toll-like receptor 5 deficienc ...... emia-reperfusion in the mouse.

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Toll-like receptor 5 deficienc ...... aemia-reperfusion in the mouse

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Bernard Waeber

http://www.w3.org/2001/XMLSchema#string

François Feihl

http://www.w3.org/2001/XMLSchema#string

Lucas Liaudet

http://www.w3.org/2001/XMLSchema#string

Na Li

http://www.w3.org/2001/XMLSchema#string

Roumen Parapanov

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P2860

Bacterial flagellin triggers cardiac innate immune responses and acute contractile dysfunction

Modulating Toll-like receptor mediated signaling by (1-->3)-beta-D-glucan rapidly induces cardioprotection

Endotoxin impairs cardiac hemodynamics by affecting loading conditions but not by reducing cardiac inotropism.

Targeted inhibition of p38 mitogen-activated protein kinase antagonizes cardiac injury and cell death following ischemia-reperfusion in vivo.

Inflammatory cytokines and postmyocardial infarction remodeling.

FoxO transcription factors promote cardiomyocyte survival upon induction of oxidative stress.

Myocardial ischemia activates an injurious innate immune signaling via cardiac heat shock protein 60 and Toll-like receptor 4.

The inflammatory response in myocardial injury, repair, and remodelling.

Peroxynitrite is a major trigger of cardiomyocyte apoptosis in vitro and in vivo.

Peroxynitrite activates ERK via Raf-1 and MEK, independently from EGF receptor and p21Ras in H9C2 cardiomyocytes.

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scholarly article

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10.1042/CS20140444

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English

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129

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inflammation

Toll-like receptor

Toll-like receptor 5 deficiency exacerbates cardiac injury and inflammation induced by myocardial ischaemia-reperfusion in the mouse.

about

P2860

P2860

Toll-like receptor 5 deficiency exacerbates cardiac injury and inflammation induced by myocardial ischaemia-reperfusion in the mouse.

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