Identification of frequent chromosomal aberrations in ductal adenocarcinoma of the pancreas by comparative genomic hybridization (CGH).
about
Genome-wide array-based comparative genomic hybridization analysis of pancreatic adenocarcinoma: identification of genetic indicators that predict patient outcomeThe reg4 gene, amplified in the early stages of pancreatic cancer development, is a promising therapeutic targetAssociation between genetic subgroups of pancreatic ductal adenocarcinoma defined by high density 500 K SNP-arrays and tumor histopathologyPD2/Paf1 depletion in pancreatic acinar cells promotes acinar-to-ductal metaplasia.Genetics and pathology of pancreatic cancer.Activated Kras and Ink4a/Arf deficiency cooperate to produce metastatic pancreatic ductal adenocarcinoma.Identification of genetic alterations in pancreatic cancer by the combined use of tissue microdissection and array-based comparative genomic hybridisation.Expression of survivin, a novel inhibitor of apoptosis and cell cycle regulatory protein, in pancreatic adenocarcinomaAmplification of the urokinase-type plasminogen activator receptor (uPAR) gene in ductal pancreatic carcinomas identifies a clinically high-risk group.Global genomic analysis of intraductal papillary mucinous neoplasms of the pancreas reveals significant molecular differences compared to ductal adenocarcinomaThe molecular and cellular heterogeneity of pancreatic ductal adenocarcinoma.Loss of expression of the SWI/SNF chromatin remodeling subunit BRG1/SMARCA4 is frequently observed in intraductal papillary mucinous neoplasms of the pancreas.Genome profiling of pancreatic adenocarcinoma.Identification of SMURF1 as a possible target for 7q21.3-22.1 amplification detected in a pancreatic cancer cell line by in-house array-based comparative genomic hybridization.Array-based comparative genomic hybridization identifies localized DNA amplifications and homozygous deletions in pancreatic cancer.High-resolution genomic and expression profiling reveals 105 putative amplification target genes in pancreatic cancerIntraductal papillary-mucinous neoplasia of the pancreas: Histopathology and molecular biologyThree synchronous, sporadic and separate periampullary and pancreatic tumors: more than a coincidence?Synchronous primary epithelial tumors of the pancreasHigh expression of RelA/p65 is associated with activation of nuclear factor-kappaB-dependent signaling in pancreatic cancer and marks a patient population with poor prognosis.Frequent loss of RUNX3 gene expression in human bile duct and pancreatic cancer cell lines.Pattern of secondary genomic changes in pancreatic tumors of Tgf alpha/Trp53+/- transgenic mice.Chromosomal and methylation alterations in sporadic and familial adenomatous polyposis-related duodenal carcinomas.
P2860
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P2860
Identification of frequent chromosomal aberrations in ductal adenocarcinoma of the pancreas by comparative genomic hybridization (CGH).
description
2000 nî lūn-bûn
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2000年の論文
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2000年学术文章
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2000年学术文章
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2000年学术文章
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2000年学术文章
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2000年学术文章
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2000年學術文章
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name
Identification of frequent chr ...... e genomic hybridization (CGH).
@en
Identification of frequent chr ...... parative genomic hybridization
@nl
type
label
Identification of frequent chr ...... e genomic hybridization (CGH).
@en
Identification of frequent chr ...... parative genomic hybridization
@nl
prefLabel
Identification of frequent chr ...... e genomic hybridization (CGH).
@en
Identification of frequent chr ...... parative genomic hybridization
@nl
P2093
P2860
P1476
Identification of frequent chr ...... e genomic hybridization (CGH).
@en
P2093
P2860
P356
10.1002/(SICI)1096-9896(200005)191:1<27::AID-PATH582>3.0.CO;2-J
P577
2000-05-01T00:00:00Z