Helicobacter pylori activates mitogen-activated protein kinase cascades and induces expression of the proto-oncogenes c-fos and c-jun.
about
Helicobacter pylori persistence: biology and diseaseRole of bacteria in oncogenesisEpigenetic regulation of DNA repair machinery in Helicobacter pylori-induced gastric carcinogenesisEchoes of a distant past: The cag pathogenicity island of Helicobacter pyloriHelicobacter pylori and interleukin-8 in gastric cancerThe Helicobacter pylori cag Pathogenicity IslandSelective inhibitors of the protein tyrosine phosphatase SHP2 block cellular motility and growth of cancer cells in vitro and in vivoToll-like receptor (TLR) 2 and TLR5, but not TLR4, are required for Helicobacter pylori-induced NF-kappa B activation and chemokine expression by epithelial cellsHelicobacter pylori induces interleukin-8 secretion by Toll-like receptor 2- and Toll-like receptor 5-dependent and -independent pathwaysHelicobacter pylori and mitogen-activated protein kinases mediate activator protein-1 (AP-1) subcomponent protein expression and DNA-binding activity in gastric epithelial cells.Helicobacter pylori CagA induces a transition from polarized to invasive phenotypes in MDCK cells.Oxidative-stress-related proteome changes in Helicobacter pylori-infected human gastric mucosa.Helicobacter pylori (H. pylori) molecular signature in conjunctival mucosa-associated lymphoid tissue (MALT) lymphomaQuantitative analysis of representative proteome components and clustering of Helicobacter pylori clinical strains.Inhibitors of Src homology-2 domain containing protein tyrosine phosphatase-2 (Shp2) based on oxindole scaffolds.Helicobacter pylori modulates lymphoepithelial cell interactions leading to epithelial cell damage through Fas/Fas ligand interactionsHelicobacter pylori environmental interactions: effect of acidic conditions on H. pylori-induced gastric mucosal interleukin-8 production.Helicobacter pylori and Hepatitis C Virus Coinfection in Egyptian Patients.Curcumin alleviates matrix metalloproteinase-3 and -9 activities during eradication of Helicobacter pylori infection in cultured cells and mice.Association of H pylori cagA and vacA genotypes and IL-8 gene polymorphisms with clinical outcome of infection in Iranian patients with gastrointestinal diseases.Proteasome particle-rich structures are widely present in human epithelial neoplasms: correlative light, confocal and electron microscopy study.Epithelial intestinal cell apoptosis induced by Helicobacter pylori depends on expression of the cag pathogenicity island phenotype.Regulation of interleukin-6 promoter activation in gastric epithelial cells infected with Helicobacter pylori.Tyrosine kinases and gastric cancer.Regulation of RANTES promoter activation in gastric epithelial cells infected with Helicobacter pyloriInterleukin-8 is the single most up-regulated gene in whole genome profiling of H. pylori exposed gastric epithelial cells.Helicobacter pylori activates the early growth response 1 protein in gastric epithelial cellsIV. Helicobacter pylori strain-specific activation of signal transduction cascades related to gastric inflammation.Helicobacter pylori-related diseases.Helicobacter pylori potentiates epithelial:mesenchymal transition in gastric cancer: links to soluble HB-EGF, gastrin and matrix metalloproteinase-7.Role of innate immunity in Helicobacter pylori-induced gastric malignancy.pathogenesis of Helicobacter pylori-induced gastric inflammation.Cyclic AMP selectively uncouples mitogen-activated protein kinase cascades from activating signalsEffect of inhibition of extracellular signal-regulated kinase 1 and 2 pathway on apoptosis and bcl-2 expression in Helicobacter pylori-infected AGS cells.Characterization of CagA variable region of Helicobacter pylori isolates from Chinese patientsAn increase in integrin-linked kinase non-canonically confers NF-κB-mediated growth advantages to gastric cancer cells by activating ERK1/2.Helicobacter bilis gamma-glutamyltranspeptidase enhances inflammatory stress response via oxidative stress in colon epithelial cells.Helicobacter pylori cag pathogenicity island (cagPAI) involved in bacterial internalization and IL-8 induced responses via NOD1- and MyD88-dependent mechanisms in human biliary epithelial cells.The Mycobacterium tuberculosis early secreted antigenic target of 6 kDa inhibits T cell interferon-γ production through the p38 mitogen-activated protein kinase pathwayFunctional and intracellular signaling differences associated with the Helicobacter pylori AlpAB adhesin from Western and East Asian strains.
P2860
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P2860
Helicobacter pylori activates mitogen-activated protein kinase cascades and induces expression of the proto-oncogenes c-fos and c-jun.
description
2000 nî lūn-bûn
@nan
2000年の論文
@ja
2000年学术文章
@wuu
2000年学术文章
@zh
2000年学术文章
@zh-cn
2000年学术文章
@zh-hans
2000年学术文章
@zh-my
2000年学术文章
@zh-sg
2000年學術文章
@yue
2000年學術文章
@zh-hant
name
Helicobacter pylori activates ...... oto-oncogenes c-fos and c-jun.
@en
Helicobacter pylori activates ...... oto-oncogenes c-fos and c-jun.
@nl
type
label
Helicobacter pylori activates ...... oto-oncogenes c-fos and c-jun.
@en
Helicobacter pylori activates ...... oto-oncogenes c-fos and c-jun.
@nl
prefLabel
Helicobacter pylori activates ...... oto-oncogenes c-fos and c-jun.
@en
Helicobacter pylori activates ...... oto-oncogenes c-fos and c-jun.
@nl
P2093
P2860
P356
P1476
Helicobacter pylori activates ...... oto-oncogenes c-fos and c-jun.
@en
P2093
T Meyer-ter-Vehn
P2860
P304
16064-16072
P356
10.1074/JBC.M000959200
P407
P577
2000-05-01T00:00:00Z