Beneficial effects of endogenous and exogenous melatonin on neural reconstruction and functional recovery in an animal model of spinal cord injury - Wikidata - awgldk - TriplyDB

Beneficial effects of endogenous and exogenous melatonin on neural reconstruction and functional recovery in an animal model of spinal cord injury

Beneficial effects of endogenous and exogenous melatonin on neural reconstruction and functional recovery in an animal model of spinal cord injury

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The Incremental Induction of Neuroprotective Properties by Multiple Therapeutic Strategies for Primary and Secondary Neural Injury Forced exercise enhances functional recovery after focal cerebral ischemia in spontaneously hypertensive rats.Melatonin as a Novel Interventional Candidate for Fragile X Syndrome with Autism Spectrum Disorder in Humans Recent advances in the pharmacologic treatment of spinal cord injury Melatonin combined with exercise cannot alleviate cerebral injury in a rat model of focal cerebral ischemia/reperfusion injury.New Prophylactic and Therapeutic Strategies for Spinal Cord Injury Autophagy in degenerating axons following spinal cord injury: evidence for autophagosome biogenesis in retraction bulbs Beneficial effects of melatonin on stroke-induced muscle atrophy in focal cerebral ischemic rats.Proteomic and bioinformatic analyses of spinal cord injury‑induced skeletal muscle atrophy in rats A review of the molecular aspects of melatonin's anti-inflammatory actions: recent insights and new perspectives.Mitochondria and chloroplasts as the original sites of melatonin synthesis: a hypothesis related to melatonin's primary function and evolution in eukaryotes.Delivery of pineal melatonin to the brain and SCN: role of canaliculi, cerebrospinal fluid, tanycytes and Virchow-Robin perivascular spaces.A review of metal-catalyzed molecular damage: protection by melatonin.Neuroprotective Effect of Simvastatin via Inducing the Autophagy on Spinal Cord Injury in the Rat Model.Reduction in traumatic brain injury-induced oxidative stress, apoptosis, and calcium entry in rat hippocampus by melatonin: Possible involvement of TRPM2 channels.Melatonin Inhibits Neural Cell Apoptosis and Promotes Locomotor Recovery via Activation of the Wnt/β-Catenin Signaling Pathway After Spinal Cord Injury.Ecto-5'-nucleotidase (CD73) attenuates inflammation after spinal cord injury by promoting macrophages/microglia M2 polarization in mice.Melatonin for the treatment of spinal cord injury

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Beneficial effects of endogenous and exogenous melatonin on neural reconstruction and functional recovery in an animal model of spinal cord injury

http://www.wikidata.org/entity/Q53525282

description

article

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im August 2011 veröffentlicher wissenschaftlicher Artikel

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wetenschappelijk artikel

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наукова стаття, опублікована в серпні 2011

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ലേഖനം

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Beneficial effects of endogeno ...... al model of spinal cord injury

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Beneficial effects of endogeno ...... al model of spinal cord injury

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Beneficial effects of endogeno ...... al model of spinal cord injury

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Beneficial effects of endogeno ...... al model of spinal cord injury

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Beneficial effects of endogeno ...... al model of spinal cord injury

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Beneficial effects of endogeno ...... al model of spinal cord injury

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J.1600-079X.2011.00925.X

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Journal of Pineal Research

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Beneficial effects of endogeno ...... al model of spinal cord injury

@en

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Je-Cheol Jeon

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Joo-Heon Kim

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Kanghui Park

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Kyu-Tae Chang

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Sang-Kil Lee

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Seunghoon Lee

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Sookyoung Park

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Yonggeun Hong

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Youngjeon Lee

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Yunkyung Hong

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Pineal Melatonin: Cell Biology of Its Synthesis and of Its Physiological Interactions*

Atrogin-1, a muscle-specific F-box protein highly expressed during muscle atrophy

Identification of ubiquitin ligases required for skeletal muscle atrophy

FoxO3 coordinately activates protein degradation by the autophagic/lysosomal and proteasomal pathways in atrophying muscle cells

A sensitive and reliable locomotor rating scale for open field testing in rats

Spinal motoneuron synaptic plasticity after axotomy in the absence of inducible nitric oxide synthase

A survey of molecular details in the human pineal gland in the light of phylogeny, structure, function and chronobiological diseases.

What do we really know about the ubiquitin-proteasome pathway in muscle atrophy?

Staining of glial fibrillary acidic protein (GFAP) in lumbar spinal cord increases following a sciatic nerve constriction injury.

TNF-alpha acts via p38 MAPK to stimulate expression of the ubiquitin ligase atrogin1/MAFbx in skeletal muscle

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107-119

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scholarly article

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10.1111/J.1600-079X.2011.00925.X

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1

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52

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2011-08-19T00:00:00Z

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21854445

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