Cobalt promotes angiogenesis via hypoxia-inducible factor and protects tubulointerstitium in the remnant kidney model. - Wikidata - awgldk - TriplyDB

Cobalt promotes angiogenesis via hypoxia-inducible factor and protects tubulointerstitium in the remnant kidney model.

Cobalt promotes angiogenesis via hypoxia-inducible factor and protects tubulointerstitium in the remnant kidney model.

http://www.wikidata.org/entity/Q53659543

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Tungsten carbide-cobalt nanoparticles induce reactive oxygen species, AKT, ERK, AP-1, NF-kB, VEGF, and angiogenesis Hypoxia promotes fibrogenesis in vivo via HIF-1 stimulation of epithelial-to-mesenchymal transition Recent advances in understanding of chronic kidney disease Hypoxia and fibrosis in chronic kidney disease: crossing at pericytes Renal tubular HIF-2α expression requires VHL inactivation and causes fibrosis and cysts Molecular mechanisms of action and therapeutic uses of pharmacological inhibitors of HIF-prolyl 4-hydroxylases for treatment of ischemic diseases Hypoxia-inducible factors in the kidney Causes of genome instability: the effect of low dose chemical exposures in modern society Cytoglobin, a novel globin, plays an antifibrotic role in the kidney Genome-wide analysis reveals adaptation to high altitudes in Tibetan sheep Engineering vascularized tissues using natural and synthetic small molecules Mitogen-activated protein 3 kinase 6 mediates angiogenic and tumorigenic effects via vascular endothelial growth factor expression Novel lnc RNA regulated by HIF-1 inhibits apoptotic cell death in the renal tubular epithelial cells under hypoxia.Silencing of hypoxia-inducible factor-1α gene attenuates chronic ischemic renal injury in two-kidney, one-clip rats.Mechanisms of metabolic memory and renal hypoxia as a therapeutic target in diabetic kidney disease.Renal tubular angiogenic dysregulation in anti-Thy1.1 glomerulonephritis.siRNA-based therapy ameliorates glomerulonephritis Hypoxia, HIF, and Associated Signaling Networks in Chronic Kidney Disease.Renal Oxygenation in the Pathophysiology of Chronic Kidney Disease.Signalling pathways involved in hypoxia-induced renal fibrosis.Protective effect of zinc preconditioning against renal ischemia reperfusion injury is dose dependent.Endothelial HIF-2 mediates protection and recovery from ischemic kidney injury.Renal protection in chronic kidney disease: hypoxia-inducible factor activation vs. angiotensin II blockade Hypoxia-inducible factor activation protects the kidney from gentamicin-induced acute injury.Hypoxic preconditioning with cobalt of bone marrow mesenchymal stem cells improves cell migration and enhances therapy for treatment of ischemic acute kidney injury.Hypoxia-inducible factor-1α contributes to the profibrotic action of angiotensin II in renal medullary interstitial cells Silencing of hypoxia-inducible factor-1α gene attenuated angiotensin II-induced renal injury in Sprague-Dawley rats.Inflammation and hypoxia linked to renal injury by CCAAT/enhancer-binding protein δ.Aberrant tubuloglomerular feedback and HIF-1α confer resistance to ischemia after subtotal nephrectomy.Recent developments in epigenetics of acute and chronic kidney diseases.Urinary hypoxia-inducible factor-1alpha levels are associated with histologic chronicity changes and renal function in patients with lupus nephritis.Interactions between HIF-1α and AMPK in the regulation of cellular hypoxia adaptation in chronic kidney disease.Peritubular capillary rarefaction: a new therapeutic target in chronic kidney disease.Improving cytocompatibility of Co28Cr6Mo by TiO2 coating: gene expression study in human endothelial cells Early detection of acute kidney injury: emerging new biomarkers.Hypoxia-inducible factor signaling in the development of tissue fibrosis Adenosine A2A receptor is a unique angiogenic target of HIF-2alpha in pulmonary endothelial cells.Renal microvascular dysfunction, hypertension and CKD progression.Renal Hypoxia in CKD; Pathophysiology and Detecting Methods.Perspective in chronic kidney disease: targeting hypoxia-inducible factor (HIF) as potential therapeutic approach.

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P2860

Cobalt promotes angiogenesis via hypoxia-inducible factor and protects tubulointerstitium in the remnant kidney model.

http://www.wikidata.org/entity/Q53659543

description

2005 nî lūn-bûn

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2005年の論文

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2005年學術文章

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name

Cobalt promotes angiogenesis v ...... m in the remnant kidney model.

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Cobalt promotes angiogenesis v ...... m in the remnant kidney model.

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Cobalt promotes angiogenesis v ...... m in the remnant kidney model.

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Cobalt promotes angiogenesis v ...... m in the remnant kidney model.

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Laboratory Investigation

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Cobalt promotes angiogenesis v ...... m in the remnant kidney model.

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Ichiro Kojima

http://www.w3.org/2001/XMLSchema#string

Julie R Ingelfinger

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Masaomi Nangaku

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Stephen Adler

http://www.w3.org/2001/XMLSchema#string

Takamoto Ohse

http://www.w3.org/2001/XMLSchema#string

Tetsuhiro Tanaka

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Toshiro Fujita

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P2860

HIFalpha targeted for VHL-mediated destruction by proline hydroxylation: implications for O2 sensing

C. elegans EGL-9 and mammalian homologs define a family of dioxygenases that regulate HIF by prolyl hydroxylation

Endothelial PAS domain protein 1 (EPAS1), a transcription factor selectively expressed in endothelial cells

Biochemical purification and pharmacological inhibition of a mammalian prolyl hydroxylase acting on hypoxia-inducible factor.

Targeting of HIF-alpha to the von Hippel-Lindau ubiquitylation complex by O2-regulated prolyl hydroxylation

Asparagine hydroxylation of the HIF transactivation domain a hypoxic switch

Lack of evidence of blood pressure-independent protection by renin-angiotensin system blockade after renal ablation

Tie2 receptor expression is stimulated by hypoxia and proinflammatory cytokines in human endothelial cells

The transcription factor EPAS-1/hypoxia-inducible factor 2alpha plays an important role in vascular remodeling

Cooperative interaction of hypoxia-inducible factor-2alpha (HIF-2alpha ) and Ets-1 in the transcriptional activation of vascular endothelial growth factor receptor-2 (Flk-1)

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1292-1307

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10.1038/LABINVEST.3700328

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16127428

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