Quercetin induces protective autophagy and apoptosis through ER stress via the p-STAT3/Bcl-2 axis in ovarian cancer.

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Novel allylated monocarbonyl analogs of curcumin induce mitotic arrest and apoptosis by reactive oxygen species-mediated endoplasmic reticulum stress and inhibition of STAT3.Quercetin prevents alcohol-induced liver injury through targeting of PI3K/Akt/nuclear factor-κB and STAT3 signaling pathway.

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Quercetin induces protective autophagy and apoptosis through ER stress via the p-STAT3/Bcl-2 axis in ovarian cancer.

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2017 nî lūn-bûn

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2017年の論文

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2017年学术文章

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2017年學術文章

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Quercetin induces protective a ...... /Bcl-2 axis in ovarian cancer.

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Quercetin induces protective a ...... /Bcl-2 axis in ovarian cancer.

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Quercetin induces protective a ...... /Bcl-2 axis in ovarian cancer.

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Quercetin induces protective a ...... /Bcl-2 axis in ovarian cancer.

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Quercetin induces protective a ...... /Bcl-2 axis in ovarian cancer.

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Quercetin induces protective a ...... /Bcl-2 axis in ovarian cancer.

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T R Zhang

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ER stress-induced cell death mechanisms

Expression and clinical significance of the autophagy proteins BECLIN 1 and LC3 in ovarian cancer

New role of JAK2/STAT3 signaling in endothelial cell oxidative stress injury and protective effect of melatonin

Autophagy and metabolism

Cell death and endoplasmic reticulum stress: disease relevance and therapeutic opportunities

Ligand-independent activation of c-Met by fibronectin and α(5)β(1)-integrin regulates ovarian cancer invasion and metastasis.

The biology of ovarian cancer: new opportunities for translation

Linking ER Stress to Autophagy: Potential Implications for Cancer Therapy.

Autophagy protects against oxaliplatin-induced cell death via ER stress and ROS in Caco-2 cells

Endoplasmic reticulum stress in endometrial cancer.

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s10495-016-1334-2

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scholarly article

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ovarian cancer

Quercetin induces protective autophagy and apoptosis through ER stress via the p-STAT3/Bcl-2 axis in ovarian cancer.

about

P2860

P2860

Quercetin induces protective autophagy and apoptosis through ER stress via the p-STAT3/Bcl-2 axis in ovarian cancer.

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