Effect of a cleavage-resistant collagen mutation on left ventricular remodeling.
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Deletion of thioredoxin-interacting protein improves cardiac inotropic reserve in the streptozotocin-induced diabetic heartCardiac function of the naked mole-rat: ecophysiological responses to working undergroundModifying the mechanics of healing infarcts: Is better the enemy of good?Myofibroblasts and the extracellular matrix network in post-myocardial infarction cardiac remodelingAttenuated hypertrophic response to pressure overload in a lamin A/C haploinsufficiency mouseCardiomyocyte hypertrophy and degradation of connexin43 through spatially restricted autocrine/paracrine heparin-binding EGFMatrix metalloproteinase-13 predominates over matrix metalloproteinase-8 as the functional interstitial collagenase in mouse atheromata.Alpha1-adrenergic receptors prevent a maladaptive cardiac response to pressure overload.Regulation of matrix biology by matrix metalloproteinases.MT1-MMP-dependent remodeling of cardiac extracellular matrix structure and function following myocardial infarctionChronic and intermittent hypoxia differentially regulate left ventricular inflammatory and extracellular matrix responsesAge-related cardiac muscle sarcopenia: Combining experimental and mathematical modeling to identify mechanisms.Reconstitution of the myocardium in regenerating newt hearts is preceded by transient deposition of extracellular matrix componentsLimiting collagen turnover via collagenase-resistance attenuates right ventricular dysfunction and fibrosis in pulmonary arterial hypertension.Transcriptomic response of Escherichia coli O157:H7 to oxidative stress.Texas 3-step decellularization protocol: looking at the cardiac extracellular matrix.Regulation of VASP phosphorylation in cardiac myocytes: differential regulation by cyclic nucleotides and modulation of protein expression in diabetic and hypertrophic heart.Extracellular matrix turnover and signaling during cardiac remodeling following MI: causes and consequences.Extracellular matrix roles during cardiac repair.Type I collagen cleavage is essential for effective fibrotic repair after myocardial infarction.Matrix metalloproteinase-9 gene deletion facilitates angiogenesis after myocardial infarction.
P2860
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P2860
Effect of a cleavage-resistant collagen mutation on left ventricular remodeling.
description
2003 nî lūn-bûn
@nan
2003年の論文
@ja
2003年学术文章
@wuu
2003年学术文章
@zh-cn
2003年学术文章
@zh-hans
2003年学术文章
@zh-my
2003年学术文章
@zh-sg
2003年學術文章
@yue
2003年學術文章
@zh
2003年學術文章
@zh-hant
name
Effect of a cleavage-resistant collagen mutation on left ventricular remodeling.
@en
Effect of a cleavage-resistant collagen mutation on left ventricular remodeling.
@nl
type
label
Effect of a cleavage-resistant collagen mutation on left ventricular remodeling.
@en
Effect of a cleavage-resistant collagen mutation on left ventricular remodeling.
@nl
prefLabel
Effect of a cleavage-resistant collagen mutation on left ventricular remodeling.
@en
Effect of a cleavage-resistant collagen mutation on left ventricular remodeling.
@nl
P2093
P1433
P1476
Effect of a cleavage-resistant collagen mutation on left ventricular remodeling.
@en
P2093
Anjali Verghese
Catherine MacGillivray
Joseph Gannon
Jun Yoshioka
Masanori Aikawa
Merry L Lindsey
Richard T Lee
Stephen M Krane
Suphichaya Muangman
P304
P356
10.1161/01.RES.0000085580.45279.60
P50
P577
2003-07-10T00:00:00Z