Evidence that Dot-dependent and -independent factors isolate the Legionella pneumophila phagosome from the endocytic network in mouse macrophages.
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RETRACTED: Molecular characterization of Legionella pneumophila-induced interleukin-8 expression in T cellsSubversion of cellular autophagosomal machinery by RNA viruses.Nutrient salvaging and metabolism by the intracellular pathogen Legionella pneumophilaLegionella dumoffii DjlA, a member of the DnaJ family, is required for intracellular growthInhibition of host vacuolar H+-ATPase activity by a Legionella pneumophila effector.Afipia felis induces uptake by macrophages directly into a nonendocytic compartment.Characterization of Legionella pneumophila pmiA, a gene essential for infectivity of protozoa and macrophages.Components of the Legionella pneumophila flagellar regulon contribute to multiple virulence traits, including lysosome avoidance and macrophage deathThe Legionella pneumophila effector VipA is an actin nucleator that alters host cell organelle trafficking.Experimental evolution of Legionella pneumophila in mouse macrophages leads to strains with altered determinants of environmental survival.Pathogenic trickery: deception of host cell processes.Genetic and phenotypic differences between Legionella pneumophila strainsBacterial interactions with the autophagic pathway.Trafficking of the Salmonella vacuole in macrophages.Type IVB secretion by intracellular pathogens.Membrane vesicles shed by Legionella pneumophila inhibit fusion of phagosomes with lysosomesAutophagy is an immediate macrophage response to Legionella pneumophila.Macrophages rapidly transfer pathogens from lipid raft vacuoles to autophagosomes.Secrets of a successful pathogen: legionella resistance to progression along the autophagic pathway.Legionella pneumophila catalase-peroxidases are required for proper trafficking and growth in primary macrophagesEnvironmental mimics and the Lvh type IVA secretion system contribute to virulence-related phenotypes of Legionella pneumophila.Cellular autophagy: surrender, avoidance and subversion by microorganisms.MavN is a Legionella pneumophila vacuole-associated protein required for efficient iron acquisition during intracellular growth.ClpP-deletion impairs the virulence of Legionella pneumophila and the optimal translocation of effector proteinsLegionella pneumophila type II protein secretion promotes virulence in the A/J mouse model of Legionnaires' disease pneumonia.Virulent Coxiella burnetii pathotypes productively infect primary human alveolar macrophagesPassage through Tetrahymena tropicalis triggers a rapid morphological differentiation in Legionella pneumophilaManipulation of rab GTPase function by intracellular bacterial pathogensMacrophages from mice with the restrictive Lgn1 allele exhibit multifactorial resistance to Legionella pneumophilaThe many forms of a pleomorphic bacterial pathogen-the developmental network of Legionella pneumophila.Ultrastructural analysis of differentiation in Legionella pneumophila.The autophagic pathway is actively modulated by phase II Coxiella burnetii to efficiently replicate in the host cell.Increases in c-Jun N-terminal kinase/stress-activated protein kinase and p38 activity in monocyte-derived macrophages following the uptake of Legionella pneumophila.Icm/dot-independent entry of Legionella pneumophila into amoeba and macrophage hosts.Legionella Effector AnkX Disrupts Host Cell Endocytic Recycling in a Phosphocholination-Dependent Manner.Stationary phase and mature infectious forms of Legionella pneumophila produce distinct viable but non-culturable cells.Rapid escape of the dot/icm mutants of Legionella pneumophila into the cytosol of mammalian and protozoan cells.The purified and recombinant Legionella pneumophila chaperonin alters mitochondrial trafficking and microfilament organization.RpoS co-operates with other factors to induce Legionella pneumophila virulence in the stationary phase.Inhibition of Akt/GSK3beta signalling pathway by Legionella pneumophila is involved in induction of T-cell apoptosis.
P2860
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P2860
Evidence that Dot-dependent and -independent factors isolate the Legionella pneumophila phagosome from the endocytic network in mouse macrophages.
description
2001 nî lūn-bûn
@nan
2001年の論文
@ja
2001年学术文章
@wuu
2001年学术文章
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2001年学术文章
@zh-hans
2001年学术文章
@zh-my
2001年学术文章
@zh-sg
2001年學術文章
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name
Evidence that Dot-dependent an ...... network in mouse macrophages.
@en
Evidence that Dot-dependent an ...... network in mouse macrophages.
@nl
type
label
Evidence that Dot-dependent an ...... network in mouse macrophages.
@en
Evidence that Dot-dependent an ...... network in mouse macrophages.
@nl
prefLabel
Evidence that Dot-dependent an ...... network in mouse macrophages.
@en
Evidence that Dot-dependent an ...... network in mouse macrophages.
@nl
P2093
P2860
P1476
Evidence that Dot-dependent an ...... network in mouse macrophages.
@en
P2093
Sturgill-Koszycki S
Swanson MS
P2860
P304
P356
10.1046/J.1462-5822.2001.00093.X
P577
2001-02-01T00:00:00Z