Tumor necrosis factor-alpha and Fas activate complementary Fas-associated death domain-dependent pathways that enhance apoptosis induced by gamma-irradiation. - Wikidata - awgldk - TriplyDB

Tumor necrosis factor-alpha and Fas activate complementary Fas-associated death domain-dependent pathways that enhance apoptosis induced by gamma-irradiation.

Tumor necrosis factor-alpha and Fas activate complementary Fas-associated death domain-dependent pathways that enhance apoptosis induced by gamma-irradiation.

http://www.wikidata.org/entity/Q54058506

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Sub-lethal irradiation of human colorectal tumor cells imparts enhanced and sustained susceptibility to multiple death receptor signaling pathways.Ionizing radiation as a response-enhancing agent for CD95-mediated apoptosis.Prognostic value of serum soluble Fas in patients with locally advanced unresectable rectal cancer receiving concurrent chemoradiotherapy Randomized phase III multi-institutional study of TNFerade biologic with fluorouracil and radiotherapy for locally advanced pancreatic cancer: final results Apoptosis and tumor resistance conferred by Par-4 Egr1 promotes growth and survival of prostate cancer cells. Identification of novel Egr1 target genes.Temperature-dependent activation of differential apoptotic pathways during cryoablation in a human prostate cancer model.Phorbol ester-induced apoptosis in prostate cancer cells via autocrine activation of the extrinsic apoptotic cascade: a key role for protein kinase C delta.Low p21Waf1/Cip1 protein level sensitizes testicular germ cell tumor cells to Fas-mediated apoptosis.Time- and dose-dependent changes of intracellular cytokine and cytokine receptor profile of Ewing tumour subpopulations under the influence of ionizing radiation.Enhanced apoptosis of glioma cell lines is achieved by co-delivering FasL-GFP and TRAIL with a complex Ad5 vector.Perillyl alcohol as a radio-/chemosensitizer in malignant glioma.Role of ceramide in mediating apoptosis of irradiated LNCaP prostate cancer cells.Gene delivery from the E3 region of replicating human adenovirus: evaluation of the E3B region.Gene delivery from the E3 region of replicating human adenovirus: evaluation of the 6.7 K/gp19 K region.Plant-derived SAC domain of PAR-4 (Prostate Apoptosis Response 4) exhibits growth inhibitory effects in prostate cancer cells Gene delivery from the E3 region of replicating human adenovirus: evaluation of the ADP region.Propapoptotic effects of NF-kappaB in LNCaP prostate cancer cells lead to serine protease activation.Caspase-dependent cytochrome c release and cell death in chick cardiomyocytes after simulated ischemia-reperfusion.Simian virus-40 infection inhibits DNA damage-induced enhancement of CD95 expression and function.

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P2860

Tumor necrosis factor-alpha and Fas activate complementary Fas-associated death domain-dependent pathways that enhance apoptosis induced by gamma-irradiation.

http://www.wikidata.org/entity/Q54058506

description

2000 nî lūn-bûn

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2000年の論文

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2000年学术文章

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2000年学术文章

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2000年学术文章

@zh-hans

2000年学术文章

@zh-my

2000年学术文章

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2000年學術文章

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2000年學術文章

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2000年學術文章

@zh-hant

name

Tumor necrosis factor-alpha an ...... induced by gamma-irradiation.

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Tumor necrosis factor-alpha an ...... induced by gamma-irradiation.

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type

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Tumor necrosis factor-alpha an ...... induced by gamma-irradiation.

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Tumor necrosis factor-alpha an ...... induced by gamma-irradiation.

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Tumor necrosis factor-alpha an ...... induced by gamma-irradiation.

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Tumor necrosis factor-alpha an ...... induced by gamma-irradiation.

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Journal of Biological Chemistry

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Tumor necrosis factor-alpha an ...... induced by gamma-irradiation.

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P2093

Gelmann EP

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Kimura K

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P2860

FADD, a novel death domain-containing protein, interacts with the death domain of Fas and initiates apoptosis

Cleavage of BID by caspase 8 mediates the mitochondrial damage in the Fas pathway of apoptosis

DFF, a heterodimeric protein that functions downstream of caspase-3 to trigger DNA fragmentation during apoptosis

TRADD-TRAF2 and TRADD-FADD interactions define two distinct TNF receptor 1 signal transduction pathways

ICE-LAP3, a novel mammalian homologue of the Caenorhabditis elegans cell death protein Ced-3 is activated during Fas- and tumor necrosis factor-induced apoptosis

Involvement of MACH, a novel MORT1/FADD-interacting protease, in Fas/APO-1- and TNF receptor-induced cell death

Cleavage of lamin A by Mch2 alpha but not CPP32: multiple interleukin 1 beta-converting enzyme-related proteases with distinct substrate recognition properties are active in apoptosis

FLICE, a novel FADD-homologous ICE/CED-3-like protease, is recruited to the CD95 (Fas/APO-1) death--inducing signaling complex

The CED-3/ICE-like protease Mch2 is activated during apoptosis and cleaves the death substrate lamin A

TNF- and cancer therapy-induced apoptosis: potentiation by inhibition of NF-kappaB

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8610-8617

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scholarly article

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10.1074/JBC.275.12.8610

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12

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10722700

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