Elevated salt intake impairs dilation of rat skeletal muscle resistance arteries via ANG II suppression.
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Restoration of cerebral vascular relaxation in renin congenic rats by introgression of the Dahl R renin gene.Angiotensin-(1-7) and low-dose angiotensin II infusion reverse salt-induced endothelial dysfunction via different mechanisms in rat middle cerebral arteries.High dietary sodium intake impairs endothelium-dependent dilation in healthy salt-resistant humans.Impaired relaxation of cerebral arteries in the absence of elevated salt intake in normotensive congenic rats carrying the Dahl salt-sensitive renin gene.Acute and chronic angiotensin-(1-7) restores vasodilation and reduces oxidative stress in mesenteric arteries of salt-fed ratsHigh-salt diet and hypertension: focus on the renin-angiotensin system.Dahl salt-sensitive rats are protected against vascular defects related to diet-induced obesityIncreased peripheral vascular disease risk progressively constrains perfusion adaptability in the skeletal muscle microcirculation.Low-dose angiotensin II infusion restores vascular function in cerebral arteries of high salt-fed rats by increasing copper/zinc superoxide dimutase expression.Time-course and mechanisms of restored vascular relaxation by reduced salt intake and angiotensin II infusion in rats fed a high-salt dietAT1 receptors prevent salt-induced vascular dysfunction in isolated middle cerebral arteries of 2 kidney-1 clip hypertensive rats.Angiotensin II maintains cerebral vascular relaxation via EGF receptor transactivation and ERK1/2.Relationships between vascular oxygen sensing mechanisms and hypertensive disease processes.The NRF2 knockout rat: a new animal model to study endothelial dysfunction, oxidant stress, and microvascular rarefaction.Introgression of the Brown Norway renin allele onto the Dahl salt-sensitive genetic background increases Cu/Zn SOD expression in cerebral arteries.Angiotensin-(1-7) Selectively Induces Relaxation and Modulates Endothelium-Dependent Dilation in Mesenteric Arteries of Salt-Fed Rats.Reduced angiotensin II levels cause generalized vascular dysfunction via oxidant stress in hamster cheek pouch arterioles.Modulation of vascular O2 responses by cytochrome 450-4A omega-hydroxylase metabolites in Dahl salt-sensitive rats.Angiotensin II AT1 receptors preserve vasodilator reactivity in skeletal muscle resistance arteries.High-salt diet impairs hypoxia-induced cAMP production and hyperpolarization in rat skeletal muscle arteries.High-salt diet depresses acetylcholine reactivity proximal to NOS activation in cerebral arteries.High-salt diet impairs vascular relaxation mechanisms in rat middle cerebral arteries.Effect of high-salt diet on NO release and superoxide production in rat aorta.Introgression of chromosome 13 in Dahl salt-sensitive genetic background restores cerebral vascular relaxation.Chronic AT(1) receptor blockade alters mechanisms mediating responses to hypoxia in rat skeletal muscle resistance arteries.High dietary salt reduces the contribution of 20-HETE to arteriolar oxygen responsiveness in skeletal muscle.Salt-induced ANG II suppression impairs the response of cerebral artery smooth muscle cells to prostacyclin.Effects of high-salt diet on CYP450-4A omega-hydroxylase expression and active tone in mesenteric resistance arteries.Enhanced skeletal muscle arteriolar reactivity to ANG II after recovery from ischemic acute renal failure.Congenic strains reveal the effect of the renin gene on skeletal muscle angiogenesis induced by electrical stimulation.Evaluation of Vascular Control Mechanisms Utilizing Video Microscopy of Isolated Resistance Arteries of Rats.Role of superoxide and angiotensin II suppression in salt-induced changes in endothelial Ca2+ signaling and NO production in rat aortaMicrovascular flow and tissue PO(2) in skeletal muscle of chronic reduced renal mass hypertensive ratsConsomic strategies to localize genomic regions related to vascular reactivity in the Dahl salt-sensitive rat
P2860
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P2860
Elevated salt intake impairs dilation of rat skeletal muscle resistance arteries via ANG II suppression.
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name
Elevated salt intake impairs d ...... teries via ANG II suppression.
@en
Elevated salt intake impairs d ...... teries via ANG II suppression.
@nl
type
label
Elevated salt intake impairs d ...... teries via ANG II suppression.
@en
Elevated salt intake impairs d ...... teries via ANG II suppression.
@nl
prefLabel
Elevated salt intake impairs d ...... teries via ANG II suppression.
@en
Elevated salt intake impairs d ...... teries via ANG II suppression.
@nl
P2860
P1476
Elevated salt intake impairs d ...... teries via ANG II suppression.
@en
P2093
J H Lombard
P2860
P304
P356
10.1152/AJPHEART.2000.278.2.H500
P577
2000-02-01T00:00:00Z