Insulin resistance in polycystic ovary syndrome: decreased expression of GLUT-4 glucose transporters in adipocytes.
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Inflammation in Polycystic Ovary Syndrome: underpinning of insulin resistance and ovarian dysfunctionPolycystic Ovary Syndrome: Important Underrecognised Cardiometabolic Risk Factor in Reproductive-Age WomenScientific Statement on the Diagnostic Criteria, Epidemiology, Pathophysiology, and Molecular Genetics of Polycystic Ovary Syndrome.Insulin resistance and the polycystic ovary syndrome revisited: an update on mechanisms and implications.Developmental programming: impact of prenatal testosterone excess on insulin sensitivity, adiposity, and free fatty acid profile in postpubertal female sheepElectrical vs manual acupuncture stimulation in a rat model of polycystic ovary syndrome: different effects on muscle and fat tissue insulin signaling.The expression of the miR-25/93/106b family of micro-RNAs in the adipose tissue of women with polycystic ovary syndromeThe Role of Metformin in Metabolic Disturbances during Pregnancy: Polycystic Ovary Syndrome and Gestational Diabetes Mellitus.Excessive insulin receptor serine phosphorylation in cultured fibroblasts and in skeletal muscle. A potential mechanism for insulin resistance in the polycystic ovary syndrome.Comprehensive assessment of expression of insulin signaling pathway components in subcutaneous adipose tissue of women with and without polycystic ovary syndrome.A paradigm for finding genes for a complex human trait: polycystic ovary syndrome and follistatin.Steroidogenic regulatory factor FOS is underexpressed in polycystic ovary syndrome (PCOS) adipose tissue and genetically associated with PCOS susceptibility.Increased expression of circulating miRNA-93 in women with polycystic ovary syndrome may represent a novel, non-invasive biomarker for diagnosisCirculatory estrogen level protects against breast cancer in obese women.miRNA-93 inhibits GLUT4 and is overexpressed in adipose tissue of polycystic ovary syndrome patients and women with insulin resistance.Correlation between Expression of Glucose Transporters in Granulosa Cells and Oocyte Quality in Women with Polycystic Ovary Syndrome.Low-Dose Dihydrotestosterone Drives Metabolic Dysfunction via Cytosolic and Nuclear Hepatic Androgen Receptor Mechanisms.The Pathogenesis of Polycystic Ovary Syndrome (PCOS): The Hypothesis of PCOS as Functional Ovarian Hyperandrogenism Revisited.Hyperandrogenism Accompanies Increased Intra-Abdominal Fat Storage in Normal Weight Polycystic Ovary Syndrome WomenIntense electroacupuncture normalizes insulin sensitivity, increases muscle GLUT4 content, and improves lipid profile in a rat model of polycystic ovary syndrome.Defects in insulin receptor signaling in vivo in the polycystic ovary syndrome (PCOS).
P2860
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P2860
Insulin resistance in polycystic ovary syndrome: decreased expression of GLUT-4 glucose transporters in adipocytes.
description
1993 nî lūn-bûn
@nan
1993年の論文
@ja
1993年学术文章
@wuu
1993年学术文章
@zh-cn
1993年学术文章
@zh-hans
1993年学术文章
@zh-my
1993年学术文章
@zh-sg
1993年學術文章
@yue
1993年學術文章
@zh
1993年學術文章
@zh-hant
name
Insulin resistance in polycyst ...... se transporters in adipocytes.
@en
Insulin resistance in polycyst ...... se transporters in adipocytes.
@nl
type
label
Insulin resistance in polycyst ...... se transporters in adipocytes.
@en
Insulin resistance in polycyst ...... se transporters in adipocytes.
@nl
prefLabel
Insulin resistance in polycyst ...... se transporters in adipocytes.
@en
Insulin resistance in polycyst ...... se transporters in adipocytes.
@nl
P2093
P2860
P921
P1476
Insulin resistance in polycyst ...... se transporters in adipocytes.
@en
P2093
P2860
P304
P356
10.1152/AJPENDO.1993.264.2.E197
P407
P433
P577
1993-02-01T00:00:00Z