about
Topical vitamin D3 and low-calcemic analogs induce thymic stromal lymphopoietin in mouse keratinocytes and trigger an atopic dermatitisLangerhans cell (LC) proliferation mediates neonatal development, homeostasis, and inflammation-associated expansion of the epidermal LC networkRetinoid X receptor ablation in adult mouse keratinocytes generates an atopic dermatitis triggered by thymic stromal lymphopoietinPhysiological and retinoid-induced proliferations of epidermis basal keratinocytes are differently controlled.Widespread negative response elements mediate direct repression by agonist-liganded glucocorticoid receptor.LRH-1/hB1F and HNF1 synergistically up-regulate hepatitis B virus gene transcription and DNA replication.Genetic and pharmacological evidence that a retinoic acid cannot be the RXR-activating ligand in mouse epidermis keratinocytes.Thymic stromal lymphopoietin overproduced by keratinocytes in mouse skin aggravates experimental asthma.Current evidence of epidermal barrier dysfunction and thymic stromal lymphopoietin in the atopic march.Molecular mechanism for the potentiation of the transcriptional activity of human liver receptor homolog 1 by steroid receptor coactivator-1.[Genetic dissection of retinoic acid function in epidermis physiology]Targeted conditional somatic mutagenesis in the mouse: temporally-controlled knock out of retinoid receptors in epidermal keratinocytes.Peroxisome-proliferator-activated receptor (PPAR)-gamma activation stimulates keratinocyte differentiation.Tissue-specific and inducible Cre-mediated recombination in the gut epithelium.Site-specific mutation of the hepatitis B virus enhancer II B1 element: effect on virus transcription and replication.HNF3 binds and activates the second enhancer, ENII, of hepatitis B virus.MicroRNA-146a alleviates chronic skin inflammation in atopic dermatitis through suppression of innate immune responses in keratinocytes.Skin thymic stromal lymphopoietin initiates Th2 responses through an orchestrated immune cascade.Induction of thymic stromal lymphopoietin expression in keratinocytes is necessary for generating an atopic dermatitis upon application of the active vitamin D3 analogue MC903 on mouse skin.62 Keratinocytic Thymic Stromal Lymphopoietin Plays an Important Role in Epicutaneous Sensitization and the Atopic March.Treatment of MCPT8DTR mice with high- or low-dose diphtheria toxin leads to differential depletion of basophils and granulocyte-macrophage progenitors.Aggravated TSLP-induced atopic dermatitis in mice lacking Dicer in adult skin keratinocytes.Disrupting the IL-36 and IL-23/IL-17 loop underlies the efficacy of calcipotriol and corticosteroid therapy for psoriasis
P50
Q28255158-3984B462-C351-4C21-936A-A0B2435F583BQ33590503-9BC643C6-5487-453F-AA45-F30FD3324F4CQ34078594-1BA2A3AA-13B2-442D-95E5-76CF6D9A05E5Q34079163-E4840A2A-EC7E-47D4-B974-CCC677A24BBAQ34178695-E7552595-2B70-4F56-9F10-1934B2DFDB4EQ34290447-2814181F-8633-47C3-BFCF-BCAABE8B8B0FQ34666841-A5B7A932-C895-498A-9D16-9009B00AF26EQ37083703-97B30F00-99F3-4986-8F2C-3F1C7C91C299Q38245203-F05738E3-EE6C-424F-8A4D-226769521731Q40555996-78A137A4-959F-467A-9D86-9CFA27FB343DQ44132660-8429467C-D5CD-4023-8542-F81756C96E56Q44664692-3E02E1C7-2717-47C6-B600-97818DDCC162Q44971546-7DDE8AD3-2098-4718-8CF0-67F6D6F7800AQ44999476-5F1628EC-9E60-49E8-B808-4F0ECB4FFE3CQ45738762-F3A6E13B-E9B6-4168-B5FB-D0CFDB9EC00BQ45772222-A0AF589D-0D16-417D-BFB9-68EFBDB1B65BQ45866593-4FB2451C-AB77-4126-A9A6-3E7076BDA474Q45897140-5F9E1898-D0A3-4122-9371-042814BC7B38Q46466229-D03F7677-5CED-4683-8B72-01E85A38B17EQ47137391-CFCFACBC-C53F-4C08-8B1A-FA34D7E39385Q47189888-C9B5B691-D8B6-40EE-BFD2-A262A0DA63E5Q54571304-9CFCF251-97E9-47D3-822C-D1FBD72FC165Q62660641-6A0DB187-D0C2-43AA-9C3E-A11B1681B8EA
P50
description
onderzoeker
@nl
researcher
@en
հետազոտող
@hy
name
Mei Li
@ast
Mei Li
@en
Mei Li
@es
Mei Li
@nl
Mei Li
@sl
type
label
Mei Li
@ast
Mei Li
@en
Mei Li
@es
Mei Li
@nl
Mei Li
@sl
prefLabel
Mei Li
@ast
Mei Li
@en
Mei Li
@es
Mei Li
@nl
Mei Li
@sl
P214
P106
P214
P31
P496
0000-0003-4225-6699
P7859
lccn-n2009183566