Interleukin-22 and interleukin-22-producing NKp44+ natural killer cells in subclinical gut inflammation in ankylosing spondylitis.
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HLA-B27 misfolding and ankylosing spondylitisRegulation of the adaptive immune system by innate lymphoid cellsReview: The interleukin-23/interleukin-17 axis in spondyloarthritis pathogenesis: Th17 and beyondThe genetic association between polymorphisms in lymphotoxin-α gene and ankylosing spondylitis susceptibility in Chinese group: A case-control study.Regulation of intestinal health and disease by innate lymphoid cellsUpdate on ankylosing spondylitis: current concepts in pathogenesis.Decreased bacterial diversity characterizes the altered gut microbiota in patients with psoriatic arthritis, resembling dysbiosis in inflammatory bowel disease.The role of the gut and microbes in the pathogenesis of spondyloarthritis.Consumption of Lactobacillus casei fermented milk prevents Salmonella reactive arthritis by modulating IL-23/IL-17 expressionMicrobiome in Inflammatory Arthritis and Human Rheumatic Diseases.Gut inflammation and microbiome in spondyloarthritis.Emerging Role of Interleukin 22 in Hepatitis B Virus Infection: a Double-edged Sword.IL-22 regulates lymphoid chemokine production and assembly of tertiary lymphoid organs.Maintaining intestinal health: the genetics and immunology of very early onset inflammatory bowel disease.Ankylosing spondylitis patients display altered dendritic cell and T cell populations that implicate pathogenic roles for the IL-23 cytokine axis and intestinal inflammationInnate lymphoid cell interactions with microbiota: implications for intestinal health and disease.Innate lymphoid cells in the initiation, regulation and resolution of inflammationAnkylosing spondylitis: from cells to genesEvidence that autophagy, but not the unfolded protein response, regulates the expression of IL-23 in the gut of patients with ankylosing spondylitis and subclinical gut inflammationThe role of innate and lymphoid IL-22-producing cells in the immunopathology of primary Sjögren's syndrome.Epigenetic and gene expression analysis of ankylosing spondylitis-associated loci implicate immune cells and the gut in the disease pathogenesis.The unusual suspects--innate lymphoid cells as novel therapeutic targets in IBD.Group 3 innate lymphoid cells: regulating host-commensal bacteria interactions in inflammation and cancer.Pathogenesis of ankylosing spondylitis - recent advances and future directions.The Bench-to-Bedside Story of IL-17 and the Therapeutic Efficacy of its Targeting in Spondyloarthritis.Pathogenesis of ligaments ossification in spondyloarthritis: insights and doubts.New insights toward the pathogenesis of ankylosing spondylitis; genetic variations and epigenetic modifications.Functional Genomics and Its Bench-to-Bedside Translation Pertaining to the Identified Susceptibility Alleles and Loci in Ankylosing Spondylitis.Innate lymphoid cells in autoimmunity: emerging regulators in rheumatic diseases.Review: Innate Lymphoid Cells: Sparking Inflammatory Rheumatic Disease?The role of T helper 17 cells in the pathogenesis of hepatitis B virus-related liver cirrhosis (Review).Dysregulation of Innate Lymphoid Cells in Common Variable Immunodeficiency.Macrophage phenotype in the subclinical gut inflammation of patients with ankylosing spondylitis.EMerging BiomARKers in Inflammatory Bowel Disease (EMBARK) study identifies fecal calprotectin, serum MMP9, and serum IL-22 as a novel combination of biomarkers for Crohn's disease activity: role of cross-sectional imaging.Infectious Agents and Inflammation: The Role of Microbiota in Autoimmune Arthritis.Differentially expressed mRNAs, lncRNAs, and miRNAs with associated co-expression and ceRNA networks in ankylosing spondylitis.The role of innate lymphoid cells in health and disease.The Effects of IL-22 on the Inflammatory Mediator Production, Proliferation, and Barrier Function of HUVECs.Type 3 innate lymphoid cells producing IL-17 and IL-22 are expanded in the gut, in the peripheral blood, synovial fluid and bone marrow of patients with ankylosing spondylitis.Role of Subclinical Gut Inflammation in the Pathogenesis of Spondyloarthritis.
P2860
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P2860
Interleukin-22 and interleukin-22-producing NKp44+ natural killer cells in subclinical gut inflammation in ankylosing spondylitis.
description
2011 nî lūn-bûn
@nan
2011年の論文
@ja
2011年学术文章
@wuu
2011年学术文章
@zh
2011年学术文章
@zh-cn
2011年学术文章
@zh-hans
2011年学术文章
@zh-my
2011年学术文章
@zh-sg
2011年學術文章
@yue
2011年學術文章
@zh-hant
name
Interleukin-22 and interleukin ...... ion in ankylosing spondylitis.
@en
Interleukin-22 and interleukin ...... ion in ankylosing spondylitis.
@nl
type
label
Interleukin-22 and interleukin ...... ion in ankylosing spondylitis.
@en
Interleukin-22 and interleukin ...... ion in ankylosing spondylitis.
@nl
prefLabel
Interleukin-22 and interleukin ...... ion in ankylosing spondylitis.
@en
Interleukin-22 and interleukin ...... ion in ankylosing spondylitis.
@nl
P2093
P2860
P356
P1476
Interleukin-22 and interleukin ...... ion in ankylosing spondylitis.
@en
P2093
Annarita Giardina
Antonina Accardo-Palumbo
Aroldo Rizzo
Francesca Raiata
Giacomo De Leo
Giovanni Triolo
Riccardo Alessandro
Sergio Peralta
Simona Principe
P2860
P304
P356
10.1002/ART.34355
P50
P577
2011-12-27T00:00:00Z