IL-17A induces MIP-1α expression in primary astrocytes via Src/MAPK/PI3K/NF-kB pathways: implications for multiple sclerosis.
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Control of autoimmune CNS inflammation by astrocytesPericytes contribute to the disruption of the cerebral endothelial barrier via increasing VEGF expression: implications for stroke.Interaction of astrocytes and T cells in physiological and pathological conditionsThe role of IL-17 in CNS diseases.Insights into the human brain proteome: Disclosing the biological meaning of protein networks in cerebrospinal fluid.IL-17 induces MIP-1α expression in primary mouse astrocytes via TRPC channel.Effectors of Th1 and Th17 cells act on astrocytes and augment their neuroinflammatory properties.Increased levels of MIP-1α in CSF and serum of ALS.Therapeutic Potential of Pien Tze Huang on Experimental Autoimmune Encephalomyelitis Rat.
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P2860
IL-17A induces MIP-1α expression in primary astrocytes via Src/MAPK/PI3K/NF-kB pathways: implications for multiple sclerosis.
description
2014 nî lūn-bûn
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2014年の論文
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2014年学术文章
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2014年学术文章
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2014年学术文章
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name
IL-17A induces MIP-1α expressi ...... ations for multiple sclerosis.
@en
IL-17A induces MIP-1α expressi ...... ations for multiple sclerosis.
@nl
type
label
IL-17A induces MIP-1α expressi ...... ations for multiple sclerosis.
@en
IL-17A induces MIP-1α expressi ...... ations for multiple sclerosis.
@nl
prefLabel
IL-17A induces MIP-1α expressi ...... ations for multiple sclerosis.
@en
IL-17A induces MIP-1α expressi ...... ations for multiple sclerosis.
@nl
P2093
P2860
P1476
IL-17A induces MIP-1α expressi ...... ations for multiple sclerosis.
@en
P2093
Honghong Yao
Hongwei Yi
Longxin Wang
Xiaodong Wu
Xinjian Zhu
P2860
P2888
P304
P356
10.1007/S11481-014-9553-1
P577
2014-07-03T00:00:00Z