MicroRNA-155 is essential for the T cell-mediated control of Helicobacter pylori infection and for the induction of chronic Gastritis and Colitis. - Wikidata - awgldk - TriplyDB

MicroRNA-155 is essential for the T cell-mediated control of Helicobacter pylori infection and for the induction of chronic Gastritis and Colitis.

MicroRNA-155 is essential for the T cell-mediated control of Helicobacter pylori infection and for the induction of chronic Gastritis and Colitis.

http://www.wikidata.org/entity/Q54351220

about

Induction of microRNA-155 is TLR- and type IV secretion system-dependent in macrophages and inhibits DNA-damage induced apoptosis Pathogens Use and Abuse MicroRNAs to Deceive the Immune System Expression and Function of miR-155 in Diseases of the Gastrointestinal Tract Gastric cancer development after the successful eradication of Helicobacter pylori Emerging roles of non-coding RNAs in gastric cancer: Pathogenesis and clinical implications Dysregulation of non-coding RNAs in gastric cancer Helicobacter pylori and microRNAs: Relation with innate immunity and progression of preneoplastic conditions Regulation of IL-17 in autoimmune diseases by transcriptional factors and microRNAs MicroRNAs in the interaction between host and bacterial pathogens MicroRNA regulation of T-lymphocyte immunity: modulation of molecular networks responsible for T-cell activation, differentiation, and development Progress and challenge of microRNA research in immunity MicroRNA Expression Is Altered in an Ovalbumin-Induced Asthma Model and Targeting miR-155 with Antagomirs Reveals Cellular Specificity ncRNA-regulated immune response and its role in inflammatory lung diseases Influence of Aspergillus fumigatus conidia viability on murine pulmonary microRNA and mRNA expression following subchronic inhalation exposure Modeling the role of peroxisome proliferator-activated receptor γ and microRNA-146 in mucosal immune responses to Clostridium difficile.Altered microRNA signatures in sputum of patients with active pulmonary tuberculosis.Highlights of the advances in basic immunology in 2011.Deregulation of MicroRNAs in Gastric Lymphomagenesis Induced in the d3Tx Mouse Model of Helicobacter pylori Infection miR-155 activates cytokine gene expression in Th17 cells by regulating the DNA-binding protein Jarid2 to relieve polycomb-mediated repression.Staphylococcal enterotoxin B-induced microRNA-155 targets SOCS1 to promote acute inflammatory lung injury.Transcriptomics identified a critical role for Th2 cell-intrinsic miR-155 in mediating allergy and antihelminth immunity.T cell IFN-γ suppression following alcohol and burn injury is independent of miRNA155.Identification of serum microRNAs as novel non-invasive biomarkers for early detection of gastric cancer.Circulating MicroRNA in inflammatory bowel disease.Conjunctival MicroRNA expression in inflammatory trachomatous scarring.Micro-RNA-155 deficiency prevents alcohol-induced serum endotoxin increase and small bowel inflammation in mice.Differential expression of microRNAs in preneoplastic gastric mucosa.Circulating microRNAs in patients with active pulmonary tuberculosis.AAV8-mediated in vivo overexpression of miR-155 enhances the protective capacity of genetically attenuated malarial parasites Mini but mighty: microRNAs in the pathobiology of periodontal disease.MicroRNA-155 is required for Mycobacterium bovis BCG-mediated apoptosis of macrophages The role of microRNAs in Helicobacter pylori pathogenesis and gastric carcinogenesis.TGF-β conditions intestinal T cells to express increased levels of miR-155, associated with down-regulation of IL-2 and itk mRNA.MicroRNA: A Bridge from H. pylori Infection to Gastritis and Gastric Cancer Development MicroRNAs in immune response and macrophage polarization.Role of miRNAs in CD4 T cell plasticity during inflammation and tolerance.Enhanced susceptibility to Citrobacter rodentium infection in microRNA-155-deficient mice Inhibition of IFN-γ-Induced Nitric Oxide Dependent Antimycobacterial Activity by miR-155 and C/EBPβ Rat mir-155 generated from the lncRNA Bic is 'hidden' in the alternate genomic assembly and reveals the existence of novel mammalian miRNAs and clusters.Acupuncture Decreases NF-κB p65, miR-155, and miR-21 and Increases miR-146a Expression in Chronic Atrophic Gastritis Rats.

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MicroRNA-155 is essential for the T cell-mediated control of Helicobacter pylori infection and for the induction of chronic Gastritis and Colitis.

http://www.wikidata.org/entity/Q54351220

description

2011 nî lūn-bûn

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2011年の論文

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2011年学术文章

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2011年学术文章

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2011年学术文章

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2011年学术文章

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2011年學術文章

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2011年學術文章

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name

MicroRNA-155 is essential for ...... chronic Gastritis and Colitis.

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MicroRNA-155 is essential for ...... chronic Gastritis and Colitis.

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MicroRNA-155 is essential for ...... chronic Gastritis and Colitis.

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MicroRNA-155 is essential for ...... chronic Gastritis and Colitis.

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MicroRNA-155 is essential for ...... chronic Gastritis and Colitis.

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MicroRNA-155 is essential for ...... chronic Gastritis and Colitis.

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JIMMUNOL.1101772

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Journal of Immunology

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Anne Müller

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Daniela B Engler

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Esther Kohler

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Manuel Koch

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Mathias Oertli

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3578-3586

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scholarly article

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10.4049/JIMMUNOL.1101772

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7

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187

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Thomas F. Meyer

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2011-08-31T00:00:00Z

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51608681

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21880981

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Helicobacter pylori