IL-22 and TNF-α represent a key cytokine combination for epidermal integrity during infection with Candida albicans. - Wikidata - awgldk - TriplyDB

IL-22 and TNF-α represent a key cytokine combination for epidermal integrity during infection with Candida albicans.

IL-22 and TNF-α represent a key cytokine combination for epidermal integrity during infection with Candida albicans.

http://www.wikidata.org/entity/Q54379262

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Mechanisms of interleukin-22's beneficial effects in acute pancreatitis How Chemotherapy Increases the Risk of Systemic Candidiasis in Cancer Patients: Current Paradigm and Future Directions Host-pathogen interactions between the human innate immune system and Candida albicans-understanding and modeling defense and evasion strategies Oropharyngeal Candidiasis in HIV Infection: Analysis of Impaired Mucosal Immune Response to Candida albicans in Mice Expressing the HIV-1 Transgene Th22 cells in allergic disease Interleukin-22 in kidney injury and regeneration.T Helper Cell Subsets in Clinical Manifestations of Psoriasis IL-22 and IDO1 affect immunity and tolerance to murine and human vaginal candidiasis Mucosal candidiasis elicits NF-κB activation, proinflammatory gene expression and localized neutrophilia in zebrafish Interleukin-22 mediates early host defense against Rhizomucor pusilluscan pathogens.Defective IL-17- and IL-22-dependent mucosal host response to Candida albicans determines susceptibility to oral candidiasis in mice expressing the HIV-1 transgene.Experimental human pneumococcal carriage augments IL-17A-dependent T-cell defence of the lung.Tailored immune responses: novel effector helper T cell subsets in protective immunity.Interferon-gamma as adjunctive immunotherapy for invasive fungal infections: a case series.Adaptive immunity to fungi.Vitamin D regulates cytokine patterns secreted by dendritic cells to promote differentiation of IL-22-producing T cells.Janus-like effects of type I interferon in autoimmune diseases.The role of pattern recognition receptors in the innate recognition of Candida albicans.Linking the Human Gut Microbiome to Inflammatory Cytokine Production Capacity.Interleukin-22 reduces lung inflammation during influenza A virus infection and protects against secondary bacterial infection.Cell-mediated immune responses to respiratory syncytial virus infection: magnitude, kinetics, and correlates with morbidity and age Interleukin-22 as a molecular adjuvant facilitates IL-17-producing CD8+ T cell responses against a HBV DNA vaccine in mice.The Th17 pathway and inflammatory diseases of the intestines, lungs, and skin Candida albicans morphogenesis and host defence: discriminating invasion from colonization.Autoimmune polyendocrinopathy candidiasis ectodermal dystrophy: known and novel aspects of the syndrome.Interplay between Candida albicans and the mammalian innate host defense.Th17 cells in immunity to Candida albicans.Human Th17 subsets.Th22 and related cytokines in inflammatory and autoimmune diseases.IL-22 in tissue-protective therapy.The role of T helper (TH)17 cells as a double-edged sword in the interplay of infection and autoimmunity with a focus on xenobiotic-induced immunomodulation.Interleukin-22: a likely target for treatment of autoimmune diseases Interplay between Candida albicans and the antimicrobial peptide armory.Defining Th-cell subsets in a classical and tissue-specific manner: Examples from the skin.Immunoresponses in dermatomycoses.Autoimmune polyendocrinopathy candidiasis ectodermal dystrophy.Immune defence against Candida fungal infections.C-type lectin receptors in the control of T helper cell differentiation.Interaction of Candida Species with the Skin.Implantable synthetic cytokine converter cells with AND-gate logic treat experimental psoriasis.

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IL-22 and TNF-α represent a key cytokine combination for epidermal integrity during infection with Candida albicans.

http://www.wikidata.org/entity/Q54379262

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2011 nî lūn-bûn

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2011年の論文

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2011年学术文章

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IL-22 and TNF-α represent a ke ...... fection with Candida albicans.

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IL-22 and TNF-α represent a ke ...... fection with Candida albicans.

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type

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IL-22 and TNF-α represent a ke ...... fection with Candida albicans.

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IL-22 and TNF-α represent a ke ...... fection with Candida albicans.

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IL-22 and TNF-α represent a ke ...... fection with Candida albicans.

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IL-22 and TNF-α represent a ke ...... fection with Candida albicans.

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European Journal of Immunology

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IL-22 and TNF-α represent a ke ...... nfection with Candida albicans

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P2093

Andrea Cavani

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Claudia Scarponi

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Davide Pennino

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Heidrun Behrendt

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Johannes Ring

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Kilian Eyerich

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Martin Mempel

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Martin Schaller

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Stefanie Eyerich

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Vera Wenzel

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P2860

Interleukin (IL)-22 and IL-17 are coexpressed by Th17 cells and cooperatively enhance expression of antimicrobial peptides

TNF-R1 signaling: a beautiful pathway

Memory IL-22-producing CD4+ T cells specific for Candida albicans are present in humans

IL-22 increases the innate immunity of tissues

Impaired T(H)17 cell differentiation in subjects with autosomal dominant hyper-IgE syndrome

Interleukin-22, a T(H)17 cytokine, mediates IL-23-induced dermal inflammation and acanthosis

Chronic mucocutaneous candidiasis in APECED or thymoma patients correlates with autoimmunity to Th17-associated cytokines.

Autoantibodies against IL-17A, IL-17F, and IL-22 in patients with chronic mucocutaneous candidiasis and autoimmune polyendocrine syndrome type I

IL-22-producing "T22" T cells account for upregulated IL-22 in atopic dermatitis despite reduced IL-17-producing TH17 T cells.

Interleukin-22 forms dimers that are recognized by two interleukin-22R1 receptor chains.

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1894-1901

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10.1002/EJI.201041197

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7

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41

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Jeanette Wagener

Claudia Traidl-Hoffmann

Carsten B Schmidt-Weber

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2011-05-20T00:00:00Z

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51021534

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21469124

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