TRPV1 receptor inhibition decreases CCL2-induced hyperalgesia.
about
The role of visceral hypersensitivity in irritable bowel syndrome: pharmacological targets and novel treatmentsHypersensitivity Induced by Activation of Spinal Cord PAR2 Receptors Is Partially Mediated by TRPV1 ReceptorsTRPV1 antagonist attenuates postoperative hypersensitivity by central and peripheral mechanisms.Peripheral inflammation affects modulation of nociceptive synaptic transmission in the spinal cord induced by N-arachidonoylphosphatidylethanolamine.A Combined Water Extract of Frankincense and Myrrh Alleviates Neuropathic Pain in Mice via Modulation of TRPV1.Sensing the heat with TRPM3.The association between the expression of PAR2 and TMEM16A and neuropathic pain.CCR2 upregulation in DRG neurons plays a crucial role in gastric hyperalgesia associated with diabetic gastropathy.
P2860
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P2860
TRPV1 receptor inhibition decreases CCL2-induced hyperalgesia.
description
2014 nî lūn-bûn
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2014年の論文
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2014年学术文章
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2014年学术文章
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2014年学术文章
@zh-hans
2014年学术文章
@zh-my
2014年学术文章
@zh-sg
2014年學術文章
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2014年學術文章
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2014年學術文章
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name
TRPV1 receptor inhibition decreases CCL2-induced hyperalgesia.
@en
TRPV1 receptor inhibition decreases CCL2-induced hyperalgesia.
@nl
type
label
TRPV1 receptor inhibition decreases CCL2-induced hyperalgesia.
@en
TRPV1 receptor inhibition decreases CCL2-induced hyperalgesia.
@nl
prefLabel
TRPV1 receptor inhibition decreases CCL2-induced hyperalgesia.
@en
TRPV1 receptor inhibition decreases CCL2-induced hyperalgesia.
@nl
P2093
P1433
P1476
TRPV1 receptor inhibition decreases CCL2-induced hyperalgesia.
@en
P2093
Diana Spicarova
Jiri Palecek
Nataliia Kalynovska
Petra Mrozkova
P356
10.1016/J.NEUROPHARM.2014.01.041
P50
P577
2014-02-01T00:00:00Z