Histone acetylation is essential for ANG-II-induced IGF-IIR gene expression in H9c2 cardiomyoblast cells and pathologically hypertensive rat heart.
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ANG II promotes IGF-IIR expression and cardiomyocyte apoptosis by inhibiting HSF1 via JNK activation and SIRT1 degradationHDAC4 mediates development of hypertension via vascular inflammation in spontaneous hypertensive rats.Protective effect of Danggui (Radix Angelicae Sinensis) on angiotensin II-induced apoptosis in H9c2 cardiomyoblast cells.Knowledge-based compact disease models identify new molecular players contributing to early-stage Alzheimer's disease.IGF-2R-Gαq signaling and cardiac hypertrophy in the low-birth-weight lamb.Effect of the angiotensin II receptor blocker valsartan on cardiac hypertrophy and myocardial histone deacetylase expression in rats with aortic constriction.Mitochondria and Oxidative Stress in the Cardiorenal Metabolic Syndrome.Doxorubicin attenuates CHIP-guarded HSF1 nuclear translocation and protein stability to trigger IGF-IIR-dependent cardiomyocyte death.Treatment with 17β-Estradiol Reduced Body Weight and the Risk of Cardiovascular Disease in a High-Fat Diet-Induced Animal Model of Obesity.Early origins of heart disease: low birth weight and the role of the insulin-like growth factor system in cardiac hypertrophy.Knowledge-Based Compact Disease Models: A Rapid Path from High-Throughput Data to Understanding Causative Mechanisms for a Complex Disease.Tanshinone IIA Prevents Leu27IGF-II-Induced Cardiomyocyte Hypertrophy Mediated by Estrogen Receptor and Subsequent Akt Activation.CREB Negatively Regulates IGF2R Gene Expression and Downstream Pathways to Inhibit Hypoxia-Induced H9c2 Cardiomyoblast Cell Death.Sanguinarine inhibits angiotensin II-induced apoptosis in H9c2 cardiac cells via restoring reactive oxygen species-mediated decreases in the mitochondrial membrane potentialNFIL3 suppresses hypoxia-induced apoptotic cell death by targeting the insulin-like growth factor 2 receptor.A Novel Systems-Biology Algorithm for the Analysis of Coordinated Protein Responses Using Quantitative ProteomicsMitochondrial ROS-induced ERK1/2 activation and HSF2-mediated AT1 R upregulation are required for doxorubicin-induced cardiotoxicity.Upregulation of the Na(+)-K(+)-2Cl(-) cotransporter 1 via histone modification in the aortas of angiotensin II-induced hypertensive rats.Anti-Apoptosis and Anti-Fibrosis Effects of Eriobotrya Japonica in Spontaneously Hypertensive Rat Hearts.
P2860
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P2860
Histone acetylation is essential for ANG-II-induced IGF-IIR gene expression in H9c2 cardiomyoblast cells and pathologically hypertensive rat heart.
description
2012 nî lūn-bûn
@nan
2012年の論文
@ja
2012年学术文章
@wuu
2012年学术文章
@zh-cn
2012年学术文章
@zh-hans
2012年学术文章
@zh-my
2012年学术文章
@zh-sg
2012年學術文章
@yue
2012年學術文章
@zh
2012年學術文章
@zh-hant
name
Histone acetylation is essenti ...... ically hypertensive rat heart.
@en
Histone acetylation is essenti ...... ically hypertensive rat heart.
@nl
type
label
Histone acetylation is essenti ...... ically hypertensive rat heart.
@en
Histone acetylation is essenti ...... ically hypertensive rat heart.
@nl
prefLabel
Histone acetylation is essenti ...... ically hypertensive rat heart.
@en
Histone acetylation is essenti ...... ically hypertensive rat heart.
@nl
P2093
P2860
P356
P1476
Histone acetylation is essenti ...... ically hypertensive rat heart.
@en
P2093
Bor-Show Tzang
Chang-Hai Tsai
Chih-Yang Huang
Chun-Hsien Chu
Fuu-Jen Tsai
Jeng-Fan Lo
Mu-Hsin Chang
Ru-Band Lu
Wei-Syun Hu
Yueh-Shan Weng
P2860
P304
P356
10.1002/JCP.22728
P577
2012-01-01T00:00:00Z