Control of Dkk-1 ameliorates chondrocyte apoptosis, cartilage destruction, and subchondral bone deterioration in osteoarthritic knees.
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Wnt signaling in cartilage development and diseases: lessons from animal studiesWnts talking with the TGF-β superfamily: WISPers about modulation of osteoarthritisInterplay between cartilage and subchondral bone contributing to pathogenesis of osteoarthritisLRP5 and LRP6 in development and diseaseInsights on Molecular Mechanisms of Chondrocytes Death in OsteoarthritisWnt7a Inhibits IL-1β Induced Catabolic Gene Expression and Prevents Articular Cartilage Damage in Experimental OsteoarthritisCo-expression of DKK-1 and Sclerostin in Subchondral Bone of the Proximal Femoral Heads from Osteoarthritic HipsDickkopf-1 (Dkk-1) in plasma and synovial fluid is inversely correlated with radiographic severity of knee osteoarthritis patients.Extracorporeal shockwave therapy in osteoporotic osteoarthritis of the knee in rats: an experiment in animals.Subchondral bone trabecular integrity predicts and changes concurrently with radiographic and magnetic resonance imaging-determined knee osteoarthritis progression.A complex interaction between Rickettsia conorii and Dickkopf-1--potential role in immune evasion mechanisms in endothelial cells.The Wnt inhibitor dickkopf-1: a link between breast cancer and bone metastases.Tight regulation of wingless-type signaling in the articular cartilage - subchondral bone biomechanical unit: transcriptomics in Frzb-knockout miceDel1 Knockout Mice Developed More Severe Osteoarthritis Associated with Increased Susceptibility of Chondrocytes to ApoptosisThe effects of oxygen tension and antiaging factor Klotho on Wnt signaling in nucleus pulposus cells.Characterization of genetically engineered mouse models carrying Col2a1-cre-induced deletions of Lrp5 and/or Lrp6.To Wnt or not to Wnt: the bone and joint health dilemmaDisc cell senescence in intervertebral disc degeneration: Causes and molecular pathways.Wnt and Rho GTPase signaling in osteoarthritis development and intervention: implications for diagnosis and therapy.The bone-cartilage unit in osteoarthritis.The osteoarthritic niche and modulation of skeletal stem cell function for regenerative medicine.Impact of inflammation on the osteoarthritic niche: implications for regenerative medicine.Bone remodelling in osteoarthritis.The coupling of bone and cartilage turnover in osteoarthritis: opportunities for bone antiresorptives and anabolics as potential treatments?WNT signaling and chondrocytes: from cell fate determination to osteoarthritis physiopathology.Association between serum and synovial fluid Dickkopf-1 levels with radiographic severity in primary knee osteoarthritis patients.Dickkopf1 destabilizes atherosclerotic plaques and promotes plaque formation by inducing apoptosis of endothelial cells through activation of ER stress.Osteoarthritis induction leads to early and temporal subchondral plate porosity in the tibial plateau of mice: an in vivo microfocal computed tomography study.MicroRNA-29a protects against glucocorticoid-induced bone loss and fragility in rats by orchestrating bone acquisition and resorption.Sclerostin is expressed in articular cartilage but loss or inhibition does not affect cartilage remodeling during aging or following mechanical injury.Effects of a glycogen synthase kinase-3β inhibitor (LiCl) on c-myc protein in intervertebral disc cells.The association between parathyroid hormone 1‑34 and the Wnt/β‑catenin signaling pathway in a rat model of osteoarthritis.The roles of canonical and non-canonical Wnt signaling in human de-differentiated articular chondrocytes.Serum levels of the bone turnover markers dickkopf-1, osteoprotegerin, and TNF-α in knee osteoarthritis patients.Chondrocyte cultures from human proximal interphalangeal finger joints.Dkk-1 promotes angiogenic responses and cartilage matrix proteinase secretion in synovial fibroblasts from osteoarthritic joints.Expression of pro-apoptotic markers is increased along the osteochondral junction in naturally occurring osteochondrosis.Dkk-1-Mediated Inhibition of Wnt Signaling in Bone Ameliorates Osteoarthritis in MiceThe Role of Wnt Pathway in the Pathogenesis of OA and Its Potential Therapeutic Implications in the Field of Regenerative Medicine
P2860
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P2860
Control of Dkk-1 ameliorates chondrocyte apoptosis, cartilage destruction, and subchondral bone deterioration in osteoarthritic knees.
description
2010 nî lūn-bûn
@nan
2010年の論文
@ja
2010年学术文章
@wuu
2010年学术文章
@zh
2010年学术文章
@zh-cn
2010年学术文章
@zh-hans
2010年学术文章
@zh-my
2010年学术文章
@zh-sg
2010年學術文章
@yue
2010年學術文章
@zh-hant
name
Control of Dkk-1 ameliorates c ...... ation in osteoarthritic knees.
@en
Control of Dkk-1 ameliorates c ...... ation in osteoarthritic knees.
@nl
type
label
Control of Dkk-1 ameliorates c ...... ation in osteoarthritic knees.
@en
Control of Dkk-1 ameliorates c ...... ation in osteoarthritic knees.
@nl
prefLabel
Control of Dkk-1 ameliorates c ...... ation in osteoarthritic knees.
@en
Control of Dkk-1 ameliorates c ...... ation in osteoarthritic knees.
@nl
P2093
P2860
P356
P1476
Control of Dkk-1 ameliorates c ...... ation in osteoarthritic knees.
@en
P2093
Ching-Jen Wang
Feng-Sheng Wang
Jih-Yang Ko
Lin-Hsiu Weng
Yi-Chih Sun
P2860
P304
P356
10.1002/ART.27357
P577
2010-05-01T00:00:00Z