Doxorubicin induces senescence and impairs function of human cardiac progenitor cells. - Wikidata - awgldk - TriplyDB

Doxorubicin induces senescence and impairs function of human cardiac progenitor cells.

Doxorubicin induces senescence and impairs function of human cardiac progenitor cells.

http://www.wikidata.org/entity/Q54449632

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The tell-tale heart: molecular and cellular responses to childhood anthracycline exposure Cardiac stem cells: biology and clinical applications Small molecule compounds that induce cellular senescence Recent Advances on Pathophysiology, Diagnostic and Therapeutic Insights in Cardiac Dysfunction Induced by Antineoplastic Drugs Effects of 5-fluorouracil on morphology, cell cycle, proliferation, apoptosis, autophagy and ROS production in endothelial cells and cardiomyocytes Doxorubicin impairs the insulin-like growth factor-1 system and causes insulin-like growth factor-1 resistance in cardiomyocytes.Cardiac Cell Senescence and Redox Signaling.Rho-Associated Kinase Inhibitor (Y-27632) Attenuates Doxorubicin-Induced Apoptosis of Human Cardiac Stem Cells The human amniotic fluid stem cell secretome effectively counteracts doxorubicin-induced cardiotoxicity.An update on cardio-oncology.Mesenchymal stem cell therapy for doxorubicin cardiomyopathy: hopes and fears Chitosan Treatment Delays the Induction of Senescence in Human Foreskin Fibroblast Strains.A mutation in the NADH-dehydrogenase subunit 2 suppresses fibroblast aging.Body Surface Area and Baseline Blood Pressure Predict Subclinical Anthracycline Cardiotoxicity in Women Treated for Early Breast Cancer.Bradykinin inhibits oxidative stress-induced senescence of endothelial progenitor cells through the B2R/AKT/RB and B2R/EGFR/RB signal pathways.Testosterone Antagonizes Doxorubicin-Induced Senescence of Cardiomyocytes.Benefits of antihypertensive medications for anthracycline- and trastuzumab-induced cardiotoxicity in patients with breast cancer: Insights from recent clinical trials.A comparison of replicative senescence and doxorubicin-induced premature senescence of vascular smooth muscle cells isolated from human aorta.MicroRNA-34a regulates doxorubicin-induced cardiotoxicity in rat.Regenerative toxicology: the role of stem cells in the development of chronic toxicities.Alternative Biomarkers for Combined Biology.p66Shc signaling is involved in stress responses elicited by anthracycline treatment of rat cardiomyoblasts.Doxorubicin upregulates CXCR4 via miR-200c/ZEB1-dependent mechanism in human cardiac mesenchymal progenitor cells.A small molecule inhibitor of PAI-1 protects against doxorubicin-induced cellular senescence.Patient-Specific Pluripotent Stem Cells in Doxorubicin Cardiotoxicity: A New Window Into Personalized Medicine.Mcl-1 regulates reactive oxygen species via NOX4 during chemotherapy-induced senescence.Signalling mechanisms underlying doxorubicin and Nox2 NADPH oxidase-induced cardiomyopathy: involvement of mitofusin-2.Oxidative Stress and Cellular Response to Doxorubicin: A Common Factor in the Complex Milieu of Anthracycline Cardiotoxicity.The Light and Shadow of Senescence and Inflammation in Cardiovascular Pathology and Regenerative Medicine.Cardiac Nonmyocyte Cell Functions and Crosstalks in Response to Cardiotoxic Drugs.Chitosan delaying human fibroblast senescence through downregulation of TGF-β signaling pathway.Daunorubicin Down-Regulates the Expression of Stem Cell Markers and Factors Involved in Stem Cell Migration and Homing in Rat Heart in Subchronic but not Acute Cardiomyopathy.Role of microRNAs in doxorubicin-induced cardiotoxicity: an overview of preclinical models and cancer patients.Therapeutic doses of doxorubicin induce premature senescence of human mesenchymal stem cells derived from menstrual blood, bone marrow and adipose tissue.Antiarrhythmic effect of growth factor-supplemented cardiac progenitor cells in chronic infarcted heart.Targeted myocardial delivery of GDF11 gene rejuvenates the aged mouse heart and enhances myocardial regeneration after ischemia-reperfusion injury.Qualitative and Quantitative Analysis of Cardiac Progenitor Cells in Cases of Myocarditis and Cardiomyopathy.Long noncoding RNA H19 mediates melatonin inhibition of premature senescence of c-kit(+) cardiac progenitor cells by promoting miR-675.Characterization of functional ion channels in human cardiac c-kit+ progenitor cells.Biology of premature ageing in survivors of cancer.

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Doxorubicin induces senescence and impairs function of human cardiac progenitor cells.

http://www.wikidata.org/entity/Q54449632

description

2013 nî lūn-bûn

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2013年の論文

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2013年学术文章

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2013年学术文章

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2013年学术文章

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2013年学术文章

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2013年學術文章

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2013年學術文章

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name

Doxorubicin induces senescence and impairs function of human cardiac progenitor cells.

@en

Doxorubicin induces senescence and impairs function of human cardiac progenitor cells.

@nl

type

label

Doxorubicin induces senescence and impairs function of human cardiac progenitor cells.

@en

Doxorubicin induces senescence and impairs function of human cardiac progenitor cells.

@nl

prefLabel

Doxorubicin induces senescence and impairs function of human cardiac progenitor cells.

@en

Doxorubicin induces senescence and impairs function of human cardiac progenitor cells.

@nl

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Basic Research in Cardiology

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Doxorubicin induces senescence and impairs function of human cardiac progenitor cells.

@en

P2093

Antonella De Angelis

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Caterina Frati

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Donato Cappetta

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Elena Piegari

http://www.w3.org/2001/XMLSchema#string

Federico Quaini

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Francesco Rossi

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Gallia Graiani

http://www.w3.org/2001/XMLSchema#string

Grazia Esposito

http://www.w3.org/2001/XMLSchema#string

Konrad Urbanek

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Liberato Berrino

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P2860

Juvenile exposure to anthracyclines impairs cardiac progenitor cell function and vascularization resulting in greater susceptibility to stress-induced myocardial injury in adult mice

Significance of adaptation mechanisms in adriamycin induced congestive heart failure

Late cardiac effects of doxorubicin therapy for acute lymphoblastic leukemia in childhood

Activation of cardiac progenitor cells reverses the failing heart senescent phenotype and prolongs lifespan

Senescent cells, tumor suppression, and organismal aging: good citizens, bad neighbors

DNA damage foci at dysfunctional telomeres

Anthracyclines: molecular advances and pharmacologic developments in antitumor activity and cardiotoxicity

Cellular senescence: when bad things happen to good cells

A DNA damage checkpoint response in telomere-initiated senescence

Cardiac stem cells in patients with ischaemic cardiomyopathy (SCIPIO): initial results of a randomised phase 1 trial

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