MEN1 intragenic deletions may represent the most prevalent somatic event in sporadic primary hyperparathyroidism.
about
MicroRNA deregulation in parathyroid tumours suggests an embryonic signature.Clinicopathological correlates of hyperparathyroidism.MicroRNA-142-3p, a novel target of tumor suppressor menin, inhibits osteosarcoma cell proliferation by down-regulation of FASN.Molecular genetics in primary hyperparathyroidism: the role of genetic tests in differential diagnosis, disease prevention strategy, and therapeutic planning. A 2017 update.MEN1 mutations and potentially MEN1-targeting miRNAs are responsible for menin deficiency in sporadic and MEN1 syndrome-associated primary hyperparathyroidism.Loss of p27 expression is associated with MEN1 gene mutations in sporadic parathyroid adenomas.
P2860
MEN1 intragenic deletions may represent the most prevalent somatic event in sporadic primary hyperparathyroidism.
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2012 nî lūn-bûn
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2012年の論文
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2012年学术文章
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name
MEN1 intragenic deletions may ...... c primary hyperparathyroidism.
@en
MEN1 intragenic deletions may ...... c primary hyperparathyroidism.
@nl
type
label
MEN1 intragenic deletions may ...... c primary hyperparathyroidism.
@en
MEN1 intragenic deletions may ...... c primary hyperparathyroidism.
@nl
prefLabel
MEN1 intragenic deletions may ...... c primary hyperparathyroidism.
@en
MEN1 intragenic deletions may ...... c primary hyperparathyroidism.
@nl
P2093
P50
P356
P1476
MEN1 intragenic deletions may ...... c primary hyperparathyroidism.
@en
P2093
Elsa Fonseca
Eva Barbosa
José Teixeira-Gomes
Maria Inês Alvelos
P304
P356
10.1530/EJE-12-0327
P577
2012-12-31T00:00:00Z