RPE cell senescence: a key contributor to age-related macular degeneration.
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Cellular and Molecular Pathology of Age-Related Macular Degeneration: Potential Role for ProteoglycansSenescent cells: a novel therapeutic target for aging and age-related diseasesGlucocorticoids induce senescence in primary human tenocytes by inhibition of sirtuin 1 and activation of the p53/p21 pathway: in vivo and in vitro evidence.Clinical strategies and animal models for developing senolytic agents.Inhibition of APE1/Ref-1 redox activity rescues human retinal pigment epithelial cells from oxidative stress and reduces choroidal neovascularization.Preliminary study on electrophysiological changes after cellular autograft in age-related macular degenerationHumanin Protects RPE Cells from Endoplasmic Reticulum Stress-Induced Apoptosis by Upregulation of Mitochondrial Glutathione.Matters of life and breath: A role for hypoxia in determining cell state.Klotho regulates retinal pigment epithelial functions and protects against oxidative stress.Cell surgery and growth factors in dry age-related macular degeneration: visual prognosis and morphological study.Dysfunctional autophagy in RPE, a contributing factor in age-related macular degeneration.Retinal pigment epithelium development, plasticity, and tissue homeostasis.Oxidative stress-induced premature senescence dysregulates VEGF and CFH expression in retinal pigment epithelial cells: Implications for Age-related Macular Degeneration.Autophagy regulates death of retinal pigment epithelium cells in age-related macular degenerationThe Mitochondrial-Derived Peptide Humanin Protects RPE Cells From Oxidative Stress, Senescence, and Mitochondrial Dysfunction.Cigarette smoke induced autophagy-impairment regulates AMD pathogenesis mechanisms in ARPE-19 cells.Protective Mechanisms of the Mitochondrial-Derived Peptide Humanin in Oxidative and Endoplasmic Reticulum Stress in RPE Cells.Cellular Senescence in Age-Related Macular Degeneration: Can Autophagy and DNA Damage Response Play a Role?Advances in Age-related Macular Degeneration Understanding and Therapy.Ischemic Retinopathies: Oxidative Stress and Inflammation.TAK1 is involved in the autophagy process in retinal pigment epithelial cells.Berberine Protects Human Retinal Pigment Epithelial Cells from Hydrogen Peroxide-Induced Oxidative Damage through Activation of AMPK.Regenerative Therapy by Suprachoroidal Cell Autograft in Dry Age-related Macular Degeneration: Preliminary In Vivo ReportPGC-1α Protects RPE Cells of the Aging Retina against Oxidative Stress-Induced Degeneration through the Regulation of Senescence and Mitochondrial Quality Control. The Significance for AMD PathogenesisOxidative Stress in Age-Related Macular Degeneration: Nrf2 as Therapeutic Target
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P2860
RPE cell senescence: a key contributor to age-related macular degeneration.
description
2012 nî lūn-bûn
@nan
2012年の論文
@ja
2012年学术文章
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2012年学术文章
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2012年学术文章
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2012年学术文章
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2012年学术文章
@zh-my
2012年学术文章
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2012年學術文章
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2012年學術文章
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name
RPE cell senescence: a key contributor to age-related macular degeneration.
@en
RPE cell senescence: a key contributor to age-related macular degeneration.
@nl
type
label
RPE cell senescence: a key contributor to age-related macular degeneration.
@en
RPE cell senescence: a key contributor to age-related macular degeneration.
@nl
prefLabel
RPE cell senescence: a key contributor to age-related macular degeneration.
@en
RPE cell senescence: a key contributor to age-related macular degeneration.
@nl
P1433
P1476
RPE cell senescence: a key contributor to age-related macular degeneration.
@en
P2093
Michael R Kozlowski
P304
P356
10.1016/J.MEHY.2012.01.018
P577
2012-01-30T00:00:00Z