NK cells are dysfunctional in human chronic myelogenous leukemia before and on imatinib treatment and in BCR-ABL-positive mice.
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Immune Effector Recovery in Chronic Myeloid Leukemia and Treatment-Free Remission.Increased proportion of mature NK cells is associated with successful imatinib discontinuation in chronic myeloid leukemia.The SCLtTAxBCR-ABL transgenic mouse model closely reflects the differential effects of dasatinib on normal and malignant hematopoiesis in chronic phase-CML patientsImpaired cytotoxicity associated with defective natural killer cell differentiation in myelodysplastic syndromes.Immunological effects of nilotinib prophylaxis after allogeneic stem cell transplantation in patients with advanced chronic myeloid leukemia or philadelphia chromosome-positive acute lymphoblastic leukemiaWhile at Rome miRNA and TRAIL do whatever BCR-ABL commands to do.Novel immune modulators used in hematology: impact on NK cellsImmunology and immunotherapy of chronic myeloid leukemia.How I determine if and when to recommend stopping tyrosine kinase inhibitor treatment for chronic myeloid leukaemia.Immunology of chronic myeloid leukemia: current concepts and future goals.Chronic Myeloid Leukemia: Immunobiology and Novel Immunotherapeutic Approaches.The Hypothesis of the Human iNKT/Innate CD8(+) T-Cell Axis Applied to Cancer: Evidence for a Deficiency in Chronic Myeloid Leukemia.Role of Distinct Natural Killer Cell Subsets in Anticancer Response.The Rho-ROCK pathway as a new pathological mechanism of innate immune subversion in chronic myeloid leukaemia.Immune modulation by molecular cancer targets and targeted therapies: Rationale for novel combination strategiesChronic myeloid leukemic cells trigger poly(ADP-ribose) polymerase-dependent inactivation and cell death in lymphocytes.Monoclonal Antibody Therapies for Hematological Malignancies: Not Just Lineage-Specific Targets.Sustained upregulation of effector natural killer cells in chronic myeloid leukemia after discontinuation of imatinib.Insulin-like growth factor-1 receptor (IGF-1R) inhibition promotes expansion of human NK cells which maintain their potent antitumor activity against Ewing sarcoma cells.
P2860
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P2860
NK cells are dysfunctional in human chronic myelogenous leukemia before and on imatinib treatment and in BCR-ABL-positive mice.
description
2011 nî lūn-bûn
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2011年の論文
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2011年学术文章
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2011年学术文章
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2011年学术文章
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2011年学术文章
@zh-hans
2011年学术文章
@zh-my
2011年学术文章
@zh-sg
2011年學術文章
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2011年學術文章
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name
NK cells are dysfunctional in ...... and in BCR-ABL-positive mice.
@en
NK cells are dysfunctional in ...... and in BCR-ABL-positive mice.
@nl
type
label
NK cells are dysfunctional in ...... and in BCR-ABL-positive mice.
@en
NK cells are dysfunctional in ...... and in BCR-ABL-positive mice.
@nl
prefLabel
NK cells are dysfunctional in ...... and in BCR-ABL-positive mice.
@en
NK cells are dysfunctional in ...... and in BCR-ABL-positive mice.
@nl
P2093
P2860
P50
P356
P1433
P1476
NK cells are dysfunctional in ...... t and in BCR-ABL-positive mice
@en
P2093
C I-U Chen
H Juergens
H T Maecker
L Kerstiens
M Schemionek
S Pscherer
W E Berdel
P2860
P2888
P304
P356
10.1038/LEU.2011.239
P577
2011-09-09T00:00:00Z