Mineralocorticoid receptor blockade confers renoprotection in preexisting chronic cyclosporine nephrotoxicity. - Wikidata - awgldk - TriplyDB

Mineralocorticoid receptor blockade confers renoprotection in preexisting chronic cyclosporine nephrotoxicity.

Mineralocorticoid receptor blockade confers renoprotection in preexisting chronic cyclosporine nephrotoxicity.

http://www.wikidata.org/entity/Q54592700

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Emerging biomarkers and metabolomics for assessing toxic nephropathy and acute kidney injury (AKI) in neonatology Renoprotective mechanisms of soy protein intake in the obese Zucker rat Increased susceptibility of aging kidney to ischemic injury: identification of candidate genes changed during aging, but corrected by caloric restriction Expression of kidney injury molecule-1 (Kim-1) in relation to necrosis and apoptosis during the early stages of Cd-induced proximal tubule injury Renal aspirin: will all patients with chronic kidney disease one day take spironolactone?Urinary chemokine (C-C motif) ligand 2 (monocyte chemotactic protein-1) as a tubular injury marker for early detection of cisplatin-induced nephrotoxicity.Kidney injury molecule-1 outperforms traditional biomarkers of kidney injury in preclinical biomarker qualification studies.Kidney injury molecule-1 expression in transplant biopsies is a sensitive measure of cell injury.KAP degradation by calpain is associated with CK2 phosphorylation and provides a novel mechanism for cyclosporine A-induced proximal tubule injury The mineralocorticoid receptor antagonist eplerenone reduces renal interstitial fibrosis after long-term cyclosporine treatment in rat: antagonizing cyclosporine nephrotoxicity Bilirubin attenuates the renal tubular injury by inhibition of oxidative stress and apoptosis.Biomarkers of immunosuppressant organ toxicity after transplantation: status, concepts and misconceptions Cell adhesion molecules in chemically-induced renal injury.New insights into the pathophysiology of cyclosporine nephrotoxicity: a role of aldosterone.Aldosterone induces mesangial cell apoptosis both in vivo and in vitro An additive effect of eplerenone to ACE inhibitor on slowing the progression of diabetic nephropathy in the db/db mice.Chronic epithelial kidney injury molecule-1 expression causes murine kidney fibrosis.Kidney Injury Molecule-1 (KIM-1): a specific and sensitive biomarker of kidney injury.Preclinical evaluation of novel urinary biomarkers of cadmium nephrotoxicity.A rapid urine test for early detection of kidney injury.Comparison of kidney injury molecule-1 and other nephrotoxicity biomarkers in urine and kidney following acute exposure to gentamicin, mercury, and chromium.Kidney injury molecule-1 is an early biomarker of cadmium nephrotoxicity Are recently reported biomarkers helpful for early and accurate diagnosis of acute kidney injury?Mechanisms of cyclosporine-induced renal cell apoptosis: a systematic review.Randomized Controlled Trial of Mineralocorticoid Receptor Blockade in Children with Chronic Kidney Allograft Nephropathy.Effects of spironolactone and losartan on diabetic nephropathy in a type 2 diabetic rat model.Aldosterone-induced TGF-beta1 expression is regulated by mitogen-activated protein kinases and activator protein-1 in mesangial cells.Adrenalectomy prevents renal ischemia-reperfusion injury.Inhibition of mineralocorticoid receptors with eplerenone alleviates short-term cyclosporin A nephrotoxicity in conscious rats.Spironolactone inhibits apoptosis in rat mesangial cells under hyperglycaemic conditions via the Wnt signalling pathway.Spironolactone ameliorates transplant vasculopathy in renal chronic transplant dysfunction in rats.Aldosterone and TGF-beta1 synergistically increase PAI-1 and decrease matrix degradation in rat renal mesangial and fibroblast cells.Does Mineralocorticoid Receptor Antagonism Prevent Calcineurin Inhibitor-Induced Nephrotoxicity?Kidney injury molecule-1 (KIM-1): a urinary biomarker and much more.Spironolactone ameliorates arterial medial calcification in uremic rats: the role of mineralocorticoid receptor signaling in vascular calcification.Aldosterone promotes proximal tubular cell apoptosis: role of oxidative stress.The effect of spironolactone on calcineurin inhibitor induced nephrotoxicity: a multicenter randomized, double-blind, clinical trial (the SPIREN trial).Renal ischemia-reperfusion injury is prevented by the mineralocorticoid receptor blocker spironolactone

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Mineralocorticoid receptor blockade confers renoprotection in preexisting chronic cyclosporine nephrotoxicity.

http://www.wikidata.org/entity/Q54592700

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2006 nî lūn-bûn

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2006年學術文章

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Mineralocorticoid receptor blo ...... c cyclosporine nephrotoxicity.

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Mineralocorticoid receptor blo ...... c cyclosporine nephrotoxicity.

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Mineralocorticoid receptor blo ...... c cyclosporine nephrotoxicity.

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Mineralocorticoid receptor blo ...... c cyclosporine nephrotoxicity.

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Mineralocorticoid receptor blo ...... c cyclosporine nephrotoxicity.

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AJPRENAL.00147.2006

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American Journal of Physiology - Renal Physiology

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Mineralocorticoid receptor blo ...... c cyclosporine nephrotoxicity.

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Cristino Cruz

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Jazmin Pérez-Rojas

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Jorge A Blanco

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Joseph V Bonventre

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Joyce Trujillo

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Norma A Bobadilla

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Norma Uribe

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Vishal S Vaidya

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Analysis of Relative Gene Expression Data Using Real-Time Quantitative PCR and the 2−ΔΔCT Method

Shedding of kidney injury molecule-1, a putative adhesion protein involved in renal regeneration

The effect of spironolactone on morbidity and mortality in patients with severe heart failure. Randomized Aldactone Evaluation Study Investigators

Renocortical mRNA expression of vasoactive factors during spironolactone protective effect in chronic cyclosporine nephrotoxicity

Mineralocorticoid receptor affects AP-1 and nuclear factor-kappab activation in angiotensin II-induced cardiac injury.

Aldosterone directly induces myocyte apoptosis through calcineurin-dependent pathways.

Eplerenone, a selective aldosterone blocker, in patients with left ventricular dysfunction after myocardial infarction.

Human kidney injury molecule-1 is a tissue and urinary tumor marker of renal cell carcinoma

CLINICAL CHARACTERISTICS OF PRIMARY ALDOSTERONISM FROM AN ANALYSIS OF 145 CASES.

Aldosterone target organ protection by eplerenone.

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10.1152/AJPRENAL.00147.2006

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