Early induction of matrix metalloproteinase-9 transduces signaling in human heart end stage failure. - Wikidata - awgldk - TriplyDB

Early induction of matrix metalloproteinase-9 transduces signaling in human heart end stage failure.

Early induction of matrix metalloproteinase-9 transduces signaling in human heart end stage failure.

http://www.wikidata.org/entity/Q54638672

about

Decellularized matrices for cardiovascular tissue engineering Cardiac matrix: a clue for future therapy GABAA receptor agonist mitigates homocysteine-induced cerebrovascular remodeling in knockout mice.H2S ameliorates oxidative and proteolytic stresses and protects the heart against adverse remodeling in chronic heart failure Sphingosine 1-Phosphate Receptors: Do They Have a Therapeutic Potential in Cardiac Fibrosis?Protease-activated receptor-1 contributes to cardiac remodeling and hypertrophy Cardiac remodeling in obesity.PAR1-dependent COX-2/PGE2 production contributes to cell proliferation via EP2 receptors in primary human cardiomyocytes Protease-activated receptors and myocardial infarction Role of mitochondrial fission and fusion in cardiomyocyte contractility.Effects of thrombin and thrombin receptor activation on cardiac function after acute myocardial infarction Cardiorenal fibrosis and dysfunction in aging: Imbalance in mediators and regulators of collagen.Cardiac dys-synchronization and arrhythmia in hyperhomocysteinemia.Homocysteine, brain natriuretic peptide and chronic heart failure: a critical review.Protease-activated receptor 1 inhibition by SCH79797 attenuates left ventricular remodeling and profibrotic activities of cardiac fibroblasts.Atrial extracellular matrix remodelling in patients with atrial fibrillation.Pleiotropic effects of factor Xa and thrombin: what to expect from novel anticoagulants.Dysregulation of Mfn2 and Drp-1 proteins in heart failure.Building a bridge to recovery: the pathophysiology of LVAD-induced reverse modeling in heart failure.Extracellular matrix-mediated cellular communication in the heart.Novel mechanisms in the pathogenesis of atrial fibrillation: practical applications.Recent clinical and experimental advances in atrial fibrillation β-adrenergic receptor stimulation transactivates protease-activated receptor 1 via matrix metalloproteinase 13 in cardiac cells.Unexpected anti-hypertrophic responses to low-level stimulation of protease-activated receptors in adult rat cardiomyocytes.Omega-3E treatment regulates matrix metalloproteinases and prevents vascular reactivity alterations in diabetic rat aorta.Direct thrombin inhibition with dabigatran attenuates pressure overload-induced cardiac fibrosis and dysfunction in mice.Hypercoagulability causes atrial fibrosis and promotes atrial fibrillation.Plasma heparin cofactor II activity is inversely associated with left atrial volume and diastolic dysfunction in humans with cardiovascular risk factors.

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P2860

Early induction of matrix metalloproteinase-9 transduces signaling in human heart end stage failure.

http://www.wikidata.org/entity/Q54638672

description

2005 nî lūn-bûn

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2005年の論文

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2005年学术文章

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2005年学术文章

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2005年学术文章

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2005年学术文章

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2005年学术文章

@zh-sg

2005年學術文章

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2005年學術文章

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2005年學術文章

@zh-hant

name

Early induction of matrix meta ...... human heart end stage failure.

@en

Early induction of matrix meta ...... human heart end stage failure.

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type

label

Early induction of matrix meta ...... human heart end stage failure.

@en

Early induction of matrix meta ...... human heart end stage failure.

@nl

prefLabel

Early induction of matrix meta ...... human heart end stage failure.

@en

Early induction of matrix meta ...... human heart end stage failure.

@nl

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J.1582-4934.2005.TB00501.X

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Journal of Cellular and Molecular Medicine

P1476

Early induction of matrix meta ...... human heart end stage failure

@en

P2093

Alexander Ovechkin

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Brooke Henderson

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Giorgio M Aru

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Karni S Moshal

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Mesia Steed

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Neetu Tyagi

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Suresh C Tyagi

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Valerie Moss

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P2860

A rapid and sensitive method for the quantitation of microgram quantities of protein utilizing the principle of protein-dye binding

Protease-activated receptor (PAR), a novel family of G protein-coupled seven trans-membrane domain receptors: activation mechanisms and physiological roles.

The zymogen of tadpole collagenase.

Structural basis of end-stage failure in ischemic cardiomyopathy in humans.

Cancer metastasis and angiogenesis: an imbalance of positive and negative regulation.

Proteinases and extracellular matrix remodeling.

Myocardial interstitial matrix metalloproteinase activity is altered by mechanical changes in LV load: interaction with the angiotensin type 1 receptor.

Real-time zymography and reverse zymography: a method for detecting activities of matrix metalloproteinases and their inhibitors using FITC-labeled collagen and casein as substrates.

Induction of oxidative stress and disintegrin metalloproteinase in human heart end-stage failure.

Attenuation of oxidative stress and remodeling by cardiac inhibitor of metalloproteinase protein transfer.

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704-713

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10.1111/J.1582-4934.2005.TB00501.X

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16202218

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