SB203580, a p38 mitogen-activated protein kinase inhibitor, suppresses the development of endometriosis by down-regulating proinflammatory cytokines and proteolytic factors in a mouse model.

SB203580, a p38 mitogen-activated protein kinase inhibitor, suppresses the development of endometriosis by down-regulating proinflammatory cytokines and proteolytic factors in a mouse model.

Item
http://www.wikidata.org/entity/Q54641749
EMMPRIN is secreted by human uterine epithelial cells in microvesicles and stimulates metalloproteinase production by human uterine fibroblast cellsLipoxin A4 suppresses the development of endometriosis in an ALX receptor-dependent manner via the p38 MAPK pathway.Inhibition of p38-MAPK signaling pathway attenuates breast cancer induced bone pain and disease progression in a murine model of cancer-induced bone pain.Effect of interleukin-1β and lipoxin A4 in human endometriotic stromal cells: Proteomic analysis.Towards an understanding of the molecular mechanism of endometriosis: unbalancing epithelial-stromal genetic conflict.Non-hormonal targets underlying endometriosis: A focus on molecular mechanisms.MAP kinases and the inflammatory signaling cascade as targets for the treatment of endometriosis?An overview of early drug development for endometriosis.Kinase signalling pathways in endometriosis: potential targets for non-hormonal therapeutics.Peripheral injection of SB203580 inhibits the inflammatory-dependent synthesis of proinflammatory cytokines in the hypothalamus.Alternative strategies to manipulate fibrocyte involvement in the fibrotic tissue response: pharmacokinetic inhibition and the feasibility of directed-adipogenic differentiation.P2X3 receptor involvement in endometriosis pain via ERK signaling pathway.Therapeutic potential of Wenshen Xiaozheng tang, a traditional Chinese medicine prescription, for treating endometriosis.Lipoxin A4 Suppresses Estrogen-Induced Epithelial-Mesenchymal Transition via ALXR-Dependent Manner in Endometriosis.Alpha-7 nicotinic acetylcholine receptor (nAChR) agonist inhibits the development of endometriosis by regulating inflammation.IGF-I stimulates ERβ and aromatase expression via IGF1R/PI3K/AKT-mediated transcriptional activation in endometriosis.Interleukin-1β stimulates the secretion of thymic stromal lymphopoietin (TSLP) from endometrioma stromal cells: possible involvement of TSLP in endometriosis.
P2860

SB203580, a p38 mitogen-activated protein kinase inhibitor, suppresses the development of endometriosis by down-regulating proinflammatory cytokines and proteolytic factors in a mouse model.

Item
http://www.wikidata.org/entity/Q54641749
description
2010 nî lūn-bûn
@nan
2010年の論文
@ja
2010年学术文章
@wuu
2010年学术文章
@zh-cn
2010年学术文章
@zh-hans
2010年学术文章
@zh-my
2010年学术文章
@zh-sg
2010年學術文章
@yue
2010年學術文章
@zh
2010年學術文章
@zh-hant
name
SB203580, a p38 mitogen-activa ...... ytic factors in a mouse model.
@en
SB203580, a p38 mitogen-activa ...... ytic factors in a mouse model.
@nl
type
label
SB203580, a p38 mitogen-activa ...... ytic factors in a mouse model.
@en
SB203580, a p38 mitogen-activa ...... ytic factors in a mouse model.
@nl
prefLabel
SB203580, a p38 mitogen-activa ...... ytic factors in a mouse model.
@en
SB203580, a p38 mitogen-activa ...... ytic factors in a mouse model.
@nl
P1433
P1476
P2093
P2860
P304
P31
P356
P407
P433
P478
P577
P5875
P698
P921
P356
P1433
P1476
SB203580, a p38 mitogen-activa ...... ytic factors in a mouse model.
@en
P2093
P2860
P304
P31
P356
P407
P433
P478
P577
P5875
P698
P921