Overexpression of VEGF-A in podocytes of adult mice causes glomerular disease. - Wikidata - awgldk - TriplyDB

Overexpression of VEGF-A in podocytes of adult mice causes glomerular disease.

Overexpression of VEGF-A in podocytes of adult mice causes glomerular disease.

http://www.wikidata.org/entity/Q54682241

about

Insulin receptor substrate-2 is expressed in kidney epithelium and up-regulated in diabetic nephropathy Pericytes, an overlooked player in vascular pathobiology Acute podocyte vascular endothelial growth factor (VEGF-A) knockdown disrupts alphaVbeta3 integrin signaling in the glomerulus.Vascular endothelial growth factor-receptor 1 inhibition aggravates diabetic nephropathy through eNOS signaling pathway in db/db mice Yi qi qing re gao attenuates podocyte injury and inhibits vascular endothelial growth factor overexpression in puromycin aminonucleoside rat model.Sarpogrelate hydrochloride ameliorates diabetic nephropathy associated with inhibition of macrophage activity and inflammatory reaction in db/db mice.Podocyte-specific VEGF-a gain of function induces nodular glomerulosclerosis in eNOS null mice.Angiogenesis and chronic kidney disease.TGF-β-activated kinase 1 is crucial in podocyte differentiation and glomerular capillary formation.Induction of podocyte VEGF164 overexpression at different stages of development causes congenital nephrosis or steroid-resistant nephrotic syndrome Exacerbation of diabetic renal alterations in mice lacking vasohibin-1.KRIT1 protein depletion modifies endothelial cell behavior via increased vascular endothelial growth factor (VEGF) signaling.GIV/girdin links vascular endothelial growth factor signaling to Akt survival signaling in podocytes independent of nephrin.Glomerular endothelial cell injury and cross talk in diabetic kidney disease Relevance of VEGF and nephrin expression in glomerular diseases.Review series: The cell biology of renal filtration.Vascular endothelial growth factor receptor 2 direct interaction with nephrin links VEGF-A signals to actin in kidney podocytes Delayed Treatment with a Small Pigment Epithelium Derived Factor (PEDF) Peptide Prevents the Progression of Diabetic Renal Injury The Accumulation of VEGFA in the Glomerular Basement Membrane and Its Relationship with Podocyte Injury and Proteinuria in Alport Syndrome.Protein-energy malnutrition during early gestation in sheep blunts fetal renal vascular and nephron development and compromises adult renal function Low-grade risk of hypercoagulable state in patients suffering from diabetes mellitus type 2 Stability and Species Specificity of Renal VEGF-A Splicing Patterns in Kidney Disease.Twenty years after ACEIs and ARBs: emerging treatment strategies for diabetic nephropathy.Diabetic angiopathy and angiogenic defects.VEGF165b overexpression restores normal glomerular water permeability in VEGF164-overexpressing adult mice.Vegfa protects the glomerular microvasculature in diabetes.Autocrine effect of vascular endothelial growth factor-A is essential for mitochondrial function in brown adipocytes.Vascular Endothelial Growth Factor C for Polycystic Kidney Diseases Protective role of small pigment epithelium-derived factor (PEDF) peptide in diabetic renal injury.Endothelial-podocyte crosstalk: the missing link between endothelial dysfunction and albuminuria in diabetes.Podocytes: the Weakest Link in Diabetic Kidney Disease?Elevated expression levels of serum insulin-like growth factor-1, tumor necrosis factor-α and vascular endothelial growth factor 165 might exacerbate type 2 diabetic nephropathy.New insights into molecular mechanisms of diabetic kidney disease.Immunopathogenesis of idiopathic nephrotic syndrome with relapse.Semaphorins in kidney development and disease: modulators of ureteric bud branching, vascular morphogenesis, and podocyte-endothelial crosstalk.Bench-to-bedside review: Ventilation-induced renal injury through systemic mediator release--just theory or a causal relationship?Cellular and molecular mechanisms of diabetic glomerulopathy.VEGF and podocytes in diabetic nephropathy.From preeclampsia to renal disease: a role of angiogenic factors and the renin-angiotensin aldosterone system?Signal transduction in podocytes--spotlight on receptor tyrosine kinases.

P2860

Q24338974-F9BB7611-FB04-45D2-87AF-80A107B5E1A0 Q28070053-44F8BB54-8FD5-4C94-8D03-A6A3B62A5376 Q31074551-6C775800-B9BE-4D95-9808-4DF02B12501A Q31159383-F5A35287-97BF-423C-8F33-D1426A13FBB1 Q33748334-0C85AABB-4457-4048-8763-0AC83D443925 Q33825220-1A987328-4FE1-4C26-8240-2620EE29A13E Q33974608-4AC80967-508F-4387-A799-0FE7552DC3F9 Q34075013-40FEA560-46E8-4AD3-8395-4A40D64F1F80 Q34103389-2F5945E8-51F5-4D7A-9150-0A6F8812C162 Q34254364-ED9DE25F-486D-4526-89AE-17FF14F46E82 Q34257934-5DB0354D-5F23-4CA0-90E0-7967C1A867F3 Q34552843-E2106C58-8DF2-445C-BAE5-50449B0BDBF1 Q35027224-16BB9CA5-A13F-424B-804F-9D616B00BA7D Q35086477-C1FA6313-266A-4213-839A-9B410BCB60CF Q35131059-46320870-6BDB-49BB-9BC0-C1AC02372D47 Q35543167-A90A60A2-97FD-426B-9C43-0B99AE49CA40 Q35562791-CC8C3598-FAD1-429D-BC92-F23C82814D7A Q35710293-C4B6A828-587A-485E-90AB-09605243EB35 Q35746380-A2EBE9C1-3654-48E2-8289-E34FADCA79F8 Q35748939-4FE37BA0-E89E-4583-B90F-C64DE35F3988 Q36059272-7BEAB811-959B-43AE-8D90-D70FD43CEF2F Q36124029-D920A47C-0FE0-4FDC-8C49-DD3AD1263E14 Q36297539-10F73720-1B2D-42FC-B1BB-DADE753A0781 Q36301365-5AF6E5B8-D11C-4A6C-B1B7-50C1EFC7B1A4 Q36312650-181098CF-739D-479C-BE36-3B978FAB6663 Q36339615-369B47A4-9EB8-472D-B791-2AC899478B33 Q36386745-DE6382E1-39C5-4626-8F4B-094AC42BD9C6 Q36417877-BBE008BE-9F77-4FE7-9E15-E07C09ECA1B3 Q37142636-EB5A8E6D-925F-4989-8BF3-61DDAD52E856 Q37251105-57852964-DA32-4C25-98AD-B59C38ADA166 Q37340690-AD37444F-3470-4A11-9784-C12210381F6E Q37567736-A3EB50EB-0166-4AC9-AA20-59396A116ACF Q37598306-F0D58D0B-34E7-46E6-8605-2F5C9F1ECA9B Q37742644-EC2B1577-3CF4-4557-B7EE-7EBB57F940EF Q37845241-32BFC8F9-8A3F-4F9C-A4B4-46B4530C85E8 Q37925155-4B92A30C-4A1B-4B0C-90E3-22120F3827CE Q38010045-B866A57A-22B9-4E1A-8ABE-2393CFC85416 Q38041671-1CAD9ABB-B5F2-4BC5-8C4F-A11989A11DA9 Q38056887-1DB38D7E-58A6-41C1-9FA2-380D7FB2E82C Q38176169-8DE0ABEA-B64E-49D0-A289-D07D3A3C4A0B

P2860

Overexpression of VEGF-A in podocytes of adult mice causes glomerular disease.

http://www.wikidata.org/entity/Q54682241

description

2010 nî lūn-bûn

@nan

2010年の論文

@ja

2010年学术文章

@wuu

2010年学术文章

@zh

2010年学术文章

@zh-cn

2010年学术文章

@zh-hans

2010年学术文章

@zh-my

2010年学术文章

@zh-sg

2010年學術文章

@yue

2010年學術文章

@zh-hant

name

Overexpression of VEGF-A in podocytes of adult mice causes glomerular disease.

@en

Overexpression of VEGF-A in podocytes of adult mice causes glomerular disease.

@nl

type

label

Overexpression of VEGF-A in podocytes of adult mice causes glomerular disease.

@en

Overexpression of VEGF-A in podocytes of adult mice causes glomerular disease.

@nl

prefLabel

Overexpression of VEGF-A in podocytes of adult mice causes glomerular disease.

@en

Overexpression of VEGF-A in podocytes of adult mice causes glomerular disease.

@nl

P1433

Q54682241-CD0CBC82-516D-41F7-829D-F2E6415F8BD1

P1476

Q54682241-558837A4-E0FC-4825-9F72-DF73180725AB

P2093

Q54682241-19408D47-AE8E-4B65-B7CA-E5354B91F8F5

Q54682241-1A6DE5C2-AD96-4AEC-A7D7-BB0CBC12A7D4

Q54682241-6D35D36E-DE39-4F97-835B-9AEDD427A6D6

Q54682241-77B46D22-7515-4818-9526-C97493CB2236

Q54682241-9A920602-5F64-4C67-A323-8D2A7046EF3D

Q54682241-DC081C4C-632B-4B0C-BBB2-D99A4738FB33

Q54682241-EFD4D2C2-724E-4CB3-A5CA-29EC1F2A0385

Q54682241-F13EE127-BF61-49A4-9027-94370D8A2795

Q54682241-F69A4C86-4917-4A76-A1AF-CB9B73639E3B

P2888

Q54682241-4D0A6BDB-B09E-49F8-903D-4312C60D0BB6

P304

Q54682241-B9D9B2AB-1AC9-48F2-BF77-E679DCEAA57B

P31

Q54682241-AC83E9A3-F064-4C5F-9557-3CD36CC467DE

P356

Q54682241-2F8D7623-FDEE-46C9-9D44-D31AB5C95A78

P407

Q54682241-20E74980-5FF6-4666-97AD-2E3C484D4DDA

P433

Q54682241-E348E3E6-4A25-492A-A5A3-3A54A6BBF2C8

P478

Q54682241-B61D1C46-23A5-4069-9A70-DB9EC5B4D9B6

P50

Q54682241-2CAF8B7F-A393-4DE1-A86E-EC4B608F84CC

P577

Q54682241-F777BAE2-601E-4E90-8166-C8995A54AD9F

P5875

Q54682241-7194A4FC-3100-4618-BA27-24887EEF7C39

P698

Q54682241-0B6A7A4F-B346-4DDD-A164-5F44184801CA

P921

Q54682241-9243197F-6EB8-4927-8977-26F1CE9ED10F

P356

P1433

Kidney International

P1476

Overexpression of VEGF-A in podocytes of adult mice causes glomerular disease.

@en

P2093

Alda Tufro

http://www.w3.org/2001/XMLSchema#string

Claudia Bertuccio

http://www.w3.org/2001/XMLSchema#string

David B Thomas

http://www.w3.org/2001/XMLSchema#string

Delma Veron

http://www.w3.org/2001/XMLSchema#string

Guillermo Villegas

http://www.w3.org/2001/XMLSchema#string

Jason Teichman

http://www.w3.org/2001/XMLSchema#string

Juan Jimenez

http://www.w3.org/2001/XMLSchema#string

Kimberly J Reidy

http://www.w3.org/2001/XMLSchema#string

Wa Shen

http://www.w3.org/2001/XMLSchema#string

P2888

P304

989-999

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

P356

10.1038/KI.2010.64

http://www.w3.org/2001/XMLSchema#string

P407

P433

11

http://www.w3.org/2001/XMLSchema#string

P478

77

http://www.w3.org/2001/XMLSchema#string

P50

Jeffrey B. Kopp

P577

2010-03-10T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P5875

43073913

http://www.w3.org/2001/XMLSchema#string

P698

20375978

http://www.w3.org/2001/XMLSchema#string

P921