about
The induction of microRNA targeting IRS-1 is involved in the development of insulin resistance under conditions of mitochondrial dysfunction in hepatocytes.Data for differentially expressed microRNAs in saturated fatty acid palmitate-treated HepG2 cellsN-Acetylated alpha-linked acidic dipeptidase expressed in rat adipocytes is localized in the insulin-responsive glucose transporter (GLUT4) intracellular compartments and involved in the insulin-stimulated GLUT4 recruitment.EHD2 interacts with the insulin-responsive glucose transporter (GLUT4) in rat adipocytes and may participate in insulin-induced GLUT4 recruitment.Proteomic analysis of cellular change involved in mitochondria-to-nucleus communication in L6 GLUT4myc myocytes.Glucose transporters and insulin action: some insights into diabetes management.MicroRNA-126 suppresses mesothelioma malignancy by targeting IRS1 and interfering with the mitochondrial functionInduction of miR-96 by Dietary Saturated Fatty Acids Exacerbates Hepatic Insulin Resistance through the Suppression of INSR and IRS-1.C1q tumor necrosis factor α-related protein isoform 5 attenuates palmitate-induced DNA fragmentation in myocytes through an AMPK-dependent mechanism.Depletion of mitochondrial DNA up-regulates the expression of MDR1 gene via an increase in mRNA stability.C1q tumor necrosis factor alpha-related protein isoform 5 is increased in mitochondrial DNA-depleted myocytes and activates AMP-activated protein kinase.MicroRNA expression analysis in the liver of high fat diet-induced obese miceDataset on the identification of differentially expressed genes by annealing control primer-based PCR in mitochondrial DNA-depleted myocytes.Implications of microRNAs in the pathogenesis of diabetes.CTRP5 ameliorates palmitate-induced apoptosis and insulin resistance through activation of AMPK and fatty acid oxidation.Induction of miR-29a by saturated fatty acids impairs insulin signaling and glucose uptake through translational repression of IRS-1 in myocytes.Exosome-derived microRNAs in cancer metabolism: possible implications in cancer diagnostics and therapy.The induction of miR-96 by mitochondrial dysfunction causes impaired glycogen synthesis through translational repression of IRS-1 in SK-Hep1 cells.Ets-2 is involved in transcriptional regulation of C1qTNF-related protein 5 in muscle cells.Regulation of the transcriptional activation of CTRP3 in chondrocytes by c-Jun.C1qTNF-related protein-6 mediates fatty acid oxidation via the activation of the AMP-activated protein kinase.Depletion of mitochondrial DNA causes impaired glucose utilization and insulin resistance in L6 GLUT4myc myocytes.Sfrp2 is a transcriptional target of SREBP-1 in mouse chondrogenic cells.Association of carboxyl esterase with facilitative glucose transporter isoform 4 (GLUT4) intracellular compartments in rat adipocytes and its possible role in insulin-induced GLUT4 recruitment.Modulation of GLUT4 and GLUT1 recycling by insulin in rat adipocytes: kinetic analysis based on the involvement of multiple intracellular compartments.Dangnyohwan improves glucose utilization and reduces insulin resistance by increasing the adipocyte-specific GLUT4 expression in Otsuka Long-Evans Tokushima Fatty rats.The hepatocyte glucose-6-phosphatase subcomponent T3: its relationship to GLUT2.Data on the decreased expression of FOXO1 by miR-1271 in HepG2 hepatocytes.Cadmium induces impaired glucose tolerance in rat by down-regulating GLUT4 expression in adipocytes.Combination gene therapy using multidrug resistance (MDR1) gene shRNA and herpes simplex virus-thymidine kinase.Data on the expression and insulin-stimulated phosphorylation of IRS-1 by miR-96 in L6-GLUT4myc myocytes.Data on the expression of PEPCK in HepG2 hepatocytes transfected with miR-195.Data on the effect of miR-15b on the expression of INSR in murine C2C12 myocytes.Genetic risk for metabolic syndrome: examination of candidate gene polymorphisms related to lipid metabolism in Japanese people.MiR-1271 upregulated by saturated fatty acid palmitate provokes impaired insulin signaling by repressing INSR and IRS-1 expression in HepG2 cells.Obesity-induced miR-15b is linked causally to the development of insulin resistance through the repression of the insulin receptor in hepatocytes.Saturated fatty acid-induced miR-195 impairs insulin signaling and glycogen metabolism in HepG2 cells.Saturated fatty acids-induced miR-424-5p aggravates insulin resistance via targeting insulin receptor in hepatocytes
P50
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P50
description
researcher
@en
wetenschapper
@nl
հետազոտող
@hy
name
Wan Lee
@ast
Wan Lee
@en
Wan Lee
@es
Wan Lee
@nl
Wan Lee
@sl
type
label
Wan Lee
@ast
Wan Lee
@en
Wan Lee
@es
Wan Lee
@nl
Wan Lee
@sl
prefLabel
Wan Lee
@ast
Wan Lee
@en
Wan Lee
@es
Wan Lee
@nl
Wan Lee
@sl
P106
P1153
7407089703
P31
P496
0000-0002-5181-2135