Overexpressed HO-1 is associated with reduced STAT3 activation in preeclampsia placenta and inhibits STAT3 phosphorylation in placental JEG-3 cells under hypoxia.

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Overexpressed HO-1 is associated with reduced STAT3 activation in preeclampsia placenta and inhibits STAT3 phosphorylation in placental JEG-3 cells under hypoxia.

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http://www.wikidata.org/entity/Q54985974

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2016 nî lūn-bûn

@nan

2016年の論文

@ja

2016年学术文章

@wuu

2016年学术文章

@zh-cn

2016年学术文章

@zh-hans

2016年学术文章

@zh-my

2016年学术文章

@zh-sg

2016年學術文章

@yue

2016年學術文章

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2016年學術文章

@zh-hant

name

Overexpressed HO-1 is associat ...... tal JEG-3 cells under hypoxia.

@en

Overexpressed HO-1 is associat ...... tal JEG-3 cells under hypoxia.

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type

Item

label

Overexpressed HO-1 is associat ...... tal JEG-3 cells under hypoxia.

@en

Overexpressed HO-1 is associat ...... tal JEG-3 cells under hypoxia.

@nl

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Overexpressed HO-1 is associat ...... tal JEG-3 cells under hypoxia.

@en

Overexpressed HO-1 is associat ...... tal JEG-3 cells under hypoxia.

@nl

P1476

Overexpressed HO-1 is associat ...... tal JEG-3 cells under hypoxia.

@en

P2093

Hong-Mei Qu

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Li-Ping Qu

http://www.w3.org/2001/XMLSchema#string

Xian-Zhen Pan

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Xiao-Yan Li

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P2860

Targeted disruption of the mouse Stat3 gene leads to early embryonic lethality

A study of placental bed spiral arteries and trophoblast invasion in normal and severe pre-eclamptic pregnancies

Heme oxygenase-1/carbon monoxide: from basic science to therapeutic applications

Roles of STAT3 in mediating the cell growth, differentiation and survival signals relayed through the IL-6 family of cytokine receptors.

Carbon monoxide induces heme oxygenase-1 to modulate STAT3 activation in endothelial cells via S-glutathionylation.

Signal transducer and activator of transcription 3 activation up-regulates interleukin-6 autocrine production: a biochemical and genetic study of established cancer cell lines and clinical isolated human cancer cells.

Interplay between vascular endothelial growth factor (VEGF) and nuclear factor erythroid 2-related factor-2 (Nrf2): implications for preeclampsia.

Different levels of IL-1α, IL-6, TNF-α, NF-κB and PPAR-γ in monocyte cultures exposed by plasma preeclampsia and normotensive pregnancy.

Differences in apoptotic susceptibility of cytotrophoblasts and syncytiotrophoblasts in normal pregnancy to those complicated with preeclampsia and intrauterine growth restriction

The role of cytokines as inflammatory mediators in preeclampsia

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597-607

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scholarly article

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10.5114/AOMS.2016.63261

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P433

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P478

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2016-10-26T00:00:00Z

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29765448

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P921

pre-eclampsia

overexpression

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5949914

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