A direct role for NKG2D/MICA interaction in villous atrophy during celiac disease.
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Transglutaminase 2 undergoes a large conformational change upon activationHuman NKG2D-ligands: cell biology strategies to ensure immune recognitionCeliac disease: prevalence, diagnosis, pathogenesis and treatmentRecent advances in coeliac diseaseIdentification and analysis of multivalent proteolytically resistant peptides from gluten: implications for celiac sprueIL-15 triggers an antiapoptotic pathway in human intraepithelial lymphocytes that is a potential new target in celiac disease-associated inflammation and lymphomagenesisAntibody-mediated blockade of IL-15 reverses the autoimmune intestinal damage in transgenic mice that overexpress IL-15 in enterocytesNewly identified genetic risk variants for celiac disease related to the immune responseThe SPINK gene family and celiac disease susceptibilityThe cross-talk between enterocytes and intraepithelial lymphocytesDietary gluten and the development of type 1 diabetesRegulating the immune system via IL-15 transpresentationTherapeutic approaches for celiac diseasePlasmacytoid dendritic cells of the gut: relevance to immunity and pathologyCeliac disease: quantity mattersNKG2D ligands as therapeutic targetsImmune development and intestinal microbiota in celiac diseaseProbiotics, prebiotics and immunomodulation of gut mucosal defences: homeostasis and immunopathologyGliadin peptide P31-43 localises to endocytic vesicles and interferes with their maturationAntibodies in celiac disease: implications beyond diagnostics.Coeliac disease: a unique model for investigating broken tolerance in autoimmunityNKG2D Ligands in Tumor Immunity: Two Sides of a CoinRole of the intestinal cytokine microenvironment in shaping the intraepithelial lymphocyte repertoireCoeliac disease and gluten sensitivityGluten sensitivity in patients with primary Sjögren's syndromeThe gluten-free diet and its current application in coeliac disease and dermatitis herpetiformisCoeliac disease and gluten-related disorders in childhoodProtective and pro-inflammatory roles of intestinal bacteriaHLA-DQB1*0201 homozygosis predisposes to severe intestinal damage in celiac diseaseCeliac disease and autoimmunity: review and controversiesReview article: safe amounts of gluten for patients with wheat allergy or coeliac diseaseProteins specifically hyperexpressed in a coeliac disease patient with aberrant T cells.A tertiary twist to the transglutaminase taleParallels between pathogens and gluten peptides in celiac sprueTetraploid and hexaploid wheat varieties reveal large differences in expression of alpha-gliadins from homoeologous Gli-2 loci.Cellular expression, trafficking, and function of two isoforms of human ULBP5/RAET1G.Enhanced prevalence of plasmatic soluble MHC class I chain-related molecule in vascular pregnancy diseasesHuman IgG1 antibodies antagonizing activating receptor NKG2D on natural killer cellsPPAR signaling pathway and cancer-related proteins are involved in celiac disease-associated tissue damage.Hepatitis C virus (HCV) evades NKG2D-dependent NK cell responses through NS5A-mediated imbalance of inflammatory cytokines.
P2860
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P2860
A direct role for NKG2D/MICA interaction in villous atrophy during celiac disease.
description
2004 nî lūn-bûn
@nan
2004年の論文
@ja
2004年学术文章
@wuu
2004年学术文章
@zh-cn
2004年学术文章
@zh-hans
2004年学术文章
@zh-my
2004年学术文章
@zh-sg
2004年學術文章
@yue
2004年學術文章
@zh
2004年學術文章
@zh-hant
name
A direct role for NKG2D/MICA interaction in villous atrophy during celiac disease.
@en
A direct role for NKG2D/MICA interaction in villous atrophy during celiac disease.
@nl
type
label
A direct role for NKG2D/MICA interaction in villous atrophy during celiac disease.
@en
A direct role for NKG2D/MICA interaction in villous atrophy during celiac disease.
@nl
prefLabel
A direct role for NKG2D/MICA interaction in villous atrophy during celiac disease.
@en
A direct role for NKG2D/MICA interaction in villous atrophy during celiac disease.
@nl
P2093
P50
P1433
P1476
A direct role for NKG2D/MICA interaction in villous atrophy during celiac disease.
@en
P2093
Christophe Cellier
Jacques Schmitz
Jean-Jacques Mention
Nassima Fodil
Seiamak Bahram
ShaoLing Zhang
Sophie Hüe
Virginie Verkarre
P304
P356
10.1016/J.IMMUNI.2004.06.018
P407
P577
2004-09-01T00:00:00Z