Egr-1 increases angiogenesis in cartilage via binding Netrin-1 receptor DCC promoter.

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Egr-1 increases angiogenesis in cartilage via binding Netrin-1 receptor DCC promoter.

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2018 nî lūn-bûn

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2018年の論文

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2018年学术文章

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Egr-1 increases angiogenesis in cartilage via binding Netrin-1 receptor DCC promoter.

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Egr-1 increases angiogenesis in cartilage via binding Netrin-1 receptor DCC promoter.

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Egr-1 increases angiogenesis in cartilage via binding Netrin-1 receptor DCC promoter.

@en

Egr-1 increases angiogenesis in cartilage via binding Netrin-1 receptor DCC promoter.

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Egr-1 increases angiogenesis in cartilage via binding Netrin-1 receptor DCC promoter.

@en

Egr-1 increases angiogenesis in cartilage via binding Netrin-1 receptor DCC promoter.

@nl

P1476

Egr-1 increases angiogenesis in cartilage via binding Netrin-1 receptor DCC promoter.

@en

P2093

Da Liu

http://www.w3.org/2001/XMLSchema#string

Jun Sheng

http://www.w3.org/2001/XMLSchema#string

Kai Jiang

http://www.w3.org/2001/XMLSchema#string

Wei Zheng

http://www.w3.org/2001/XMLSchema#string

Xia Kang

http://www.w3.org/2001/XMLSchema#string

Ying Chen

http://www.w3.org/2001/XMLSchema#string

P2860

Functional genomics in osteoarthritis: Past, present, and future

Egr-1 inhibits the expression of extracellular matrix genes in chondrocytes by TNFalpha-induced MEK/ERK signalling

BRN2 is a transcriptional repressor of CDH13 (T-cadherin) in melanoma cells

C-reactive protein can upregulate VEGF expression to promote ADSC-induced angiogenesis by activating HIF-1α via CD64/PI3k/Akt and MAPK/ERK signaling pathways.

The role of vascular endothelial growth factor in angiogenesis.

Angiogenesis and nerve growth factor at the osteochondral junction in rheumatoid arthritis and osteoarthritis.

Netrin-1-mediated axon outgrowth requires deleted in colorectal cancer-dependent MAPK activation.

Role of vascular endothelial growth factor in regulation of physiological angiogenesis.

Netrin-1 induces angiogenesis via a DCC-dependent ERK1/2-eNOS feed-forward mechanism

Egr-1 activation by cancer-derived extracellular vesicles promotes endothelial cell migration via ERK1/2 and JNK signaling pathways

P2888

s13018-018-0826-x

P304

125

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P31

scholarly article

P356

10.1186/S13018-018-0826-X

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P433

http://www.w3.org/2001/XMLSchema#string

P478

http://www.w3.org/2001/XMLSchema#string

P577

2018-05-29T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P6179

1104290009

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P698

29843768

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P921

cartilage

P932

5975438

http://www.w3.org/2001/XMLSchema#string