Targeting microthrombosis and neuroinflammation with vepoloxamer for therapeutic neuroprotection after traumatic brain injury.

about

Targeting microthrombosis and neuroinflammation with vepoloxamer for therapeutic neuroprotection after traumatic brain injury.

Item

http://www.wikidata.org/entity/Q55072970

description

2018 nî lūn-bûn

@nan

2018年の論文

@ja

2018年学术文章

@wuu

2018年学术文章

@zh-cn

2018年学术文章

@zh-hans

2018年学术文章

@zh-my

2018年学术文章

@zh-sg

2018年學術文章

@yue

2018年學術文章

@zh

2018年學術文章

@zh-hant

name

Targeting microthrombosis and ...... after traumatic brain injury.

@en

Targeting microthrombosis and ...... after traumatic brain injury.

@nl

type

Item

label

Targeting microthrombosis and ...... after traumatic brain injury.

@en

Targeting microthrombosis and ...... after traumatic brain injury.

@nl

prefLabel

Targeting microthrombosis and ...... after traumatic brain injury.

@en

Targeting microthrombosis and ...... after traumatic brain injury.

@nl

P1476

Targeting microthrombosis and ...... after traumatic brain injury.

@en

P2093

Asim Mahmood

http://www.w3.org/2001/XMLSchema#string

Li Zhang

http://www.w3.org/2001/XMLSchema#string

Zheng Gang Zhang

http://www.w3.org/2001/XMLSchema#string

P2860

Traumatic brain injury causes platelet adenosine diphosphate and arachidonic acid receptor inhibition independent of hemorrhagic shock in humans and rats.

Differential effects of commercial-grade and purified poloxamer 188 on renal function

Poloxamer 188 protects neurons against ischemia/reperfusion injury through preserving integrity of cell membranes and blood brain barrier.

Coagulopathy in traumatic brain injury.

Poloxamer 188 (p188) as a membrane resealing reagent in biomedical applications.

In vivo temporal and spatial profile of leukocyte adhesion and migration after experimental traumatic brain injury in mice.

Endothelial Cell-Derived von Willebrand Factor Is the Major Determinant That Mediates von Willebrand Factor-Dependent Acute Ischemic Stroke by Promoting Postischemic Thrombo-Inflammation.

Kollidon VA64, a membrane-resealing agent, reduces histopathology and improves functional outcome after controlled cortical impact in mice.

The pharmacokinetics and pharmacodynamics of Kollidon VA64 dissociate its protective effects from membrane resealing after controlled cortical impact in mice.

Delayed thrombosis after traumatic brain injury in rats.

P304

413-414

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

P356

10.4103/1673-5374.228717

http://www.w3.org/2001/XMLSchema#string

P407

English

P433

http://www.w3.org/2001/XMLSchema#string

P478

http://www.w3.org/2001/XMLSchema#string

P50

Ye Xiong

Michael Chopp

P577

2018-03-01T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P698

29623919

http://www.w3.org/2001/XMLSchema#string

P921

neuronitis

P932

5900497

http://www.w3.org/2001/XMLSchema#string