Effects of the combination of As2O3 and AZT on proliferation inhibition and apoptosis induction of hepatoma HepG2 cells following silencing of Egr-1.

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Effects of the combination of As2O3 and AZT on proliferation inhibition and apoptosis induction of hepatoma HepG2 cells following silencing of Egr-1.

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2018 nî lūn-bûn

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2018年の論文

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2018年学术文章

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2018年学术文章

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2018年学术文章

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2018年学术文章

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2018年學術文章

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Effects of the combination of ...... following silencing of Egr-1.

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Effects of the combination of ...... following silencing of Egr-1.

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Item

label

Effects of the combination of ...... following silencing of Egr-1.

@en

Effects of the combination of ...... following silencing of Egr-1.

@nl

prefLabel

Effects of the combination of ...... following silencing of Egr-1.

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Effects of the combination of ...... following silencing of Egr-1.

@nl

P1476

Effects of the combination of ...... following silencing of Egr-1.

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P2093

Che Chen

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Chuan Zhao

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Li Han

http://www.w3.org/2001/XMLSchema#string

Mei Wang

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Yongjuan Liang

http://www.w3.org/2001/XMLSchema#string

Yu Liu

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P2860

A perspective on cancer cell metastasis

Retinoic acid and arsenic trioxide for acute promyelocytic leukemia.

A mitogen-activated protein kinase/extracellular signal-regulated kinase kinase (MEK)-dependent transcriptional program controls activation of the early growth response 1 (EGR1) gene during amino acid limitation

Identification and characterization of mitochondrial factors modulating thymidine kinase 2 activity.

All-trans retinoic acid/As2O3 combination yields a high quality remission and survival in newly diagnosed acute promyelocytic leukemia

Cell cycle arrest and apoptotic cell death in cultured human gastric carcinoma cells mediated by arsenic trioxide.

Phase 1 trial and pharmacokinetic study of arsenic trioxide in children and adolescents with refractory or relapsed acute leukemia, including acute promyelocytic leukemia or lymphoma.

Mechanisms of zidovudine-induced mitochondrial toxicity and myopathy.

The transcription factor Egr1 is a direct regulator of multiple tumor suppressors including TGFbeta1, PTEN, p53, and fibronectin.

Is EGR1 a potential target for prostate cancer therapy?

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3293-3301

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scholarly article

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10.2147/OTT.S155169

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English

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2018-06-01T00:00:00Z

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29910624

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5987793

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