Gray matter changes in asymptomatic C9orf72 and GRN mutation carriers.

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Gray matter changes in asymptomatic C9orf72 and GRN mutation carriers.

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2018 nî lūn-bûn

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2018年の論文

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2018年学术文章

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2018年学术文章

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2018年学术文章

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2018年学术文章

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2018年学术文章

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2018年学术文章

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2018年學術文章

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2018年學術文章

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name

Gray matter changes in asymptomatic C9orf72 and GRN mutation carriers.

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Gray matter changes in asymptomatic C9orf72 and GRN mutation carriers.

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label

Gray matter changes in asymptomatic C9orf72 and GRN mutation carriers.

@en

Gray matter changes in asymptomatic C9orf72 and GRN mutation carriers.

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Gray matter changes in asymptomatic C9orf72 and GRN mutation carriers.

@en

Gray matter changes in asymptomatic C9orf72 and GRN mutation carriers.

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P1476

Gray matter changes in asymptomatic C9orf72 and GRN mutation carriers.

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P2093

Adrienne Buller

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Claudia Jacova

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Dana Wittenberg

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Emma Dowds

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Ging-Yuek R Hsiung

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Howard H Feldman

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Ian R Mackenzie

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Karteek Popuri

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Mahadev Bhalla

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Mirza Faisal Beg

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P2860

Classification of primary progressive aphasia and its variants

Sensitivity of revised diagnostic criteria for the behavioural variant of frontotemporal dementia

Characterization of frontotemporal dementia and/or amyotrophic lateral sclerosis associated with the GGGGCC repeat expansion in C9ORF72

Neuroimaging signatures of frontotemporal dementia genetics: C9ORF72, tau, progranulin and sporadics

Frontotemporal dementia with the C9ORF72 hexanucleotide repeat expansion: clinical, neuroanatomical and neuropathological features

Expanded GGGGCC hexanucleotide repeat in noncoding region of C9ORF72 causes chromosome 9p-linked FTD and ALS

A hexanucleotide repeat expansion in C9ORF72 is the cause of chromosome 9p21-linked ALS-FTD

“Mini-mental state”

Cortical Surface-Based Analysis

Null mutations in progranulin cause ubiquitin-positive frontotemporal dementia linked to chromosome 17q21

P304

591-598

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scholarly article

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10.1016/J.NICL.2018.02.017

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P478

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Rosa Rademakers

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2018-02-17T00:00:00Z

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P698

29845007

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5964622

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