about
Functional disturbances in the striatum by region-specific ablation of NMDA receptors.Distinct profiles of anxiety and dysphoria during spontaneous withdrawal from acute morphine exposure.Nicotine response genetics in the zebrafish.Similar neurons, opposite adaptations: psychostimulant experience differentially alters firing properties in accumbens core versus shell.Mechanisms underlying the activation of G-protein-gated inwardly rectifying K+ (GIRK) channels by the novel anxiolytic drug, ML297.Putative kappa opioid heteromers as targets for developing analgesics free of adverse effects.Cocaine experience controls bidirectional synaptic plasticity in the nucleus accumbens.Spontaneous locomotor activity correlates with the degranulation of mast cells in the meninges rather than in the thalamus: disruptive effect of cocaine.An embryonic culture system for the investigation of striatal medium spiny neuron dendritic spine development and plasticitySynaptic adaptations in the nucleus accumbens caused by experiences linked to relapse.Reversal of morphine-induced cell-type-specific synaptic plasticity in the nucleus accumbens shell blocks reinstatement.Behavioral and structural responses to chronic cocaine require a feedforward loop involving ΔFosB and calcium/calmodulin-dependent protein kinase II in the nucleus accumbens shell.Biological substrates of reward and aversion: a nucleus accumbens activity hypothesis.Neuroplasticity in the mesolimbic dopamine system and cocaine addictionCocaine and Amphetamine Induce Overlapping but Distinct Patterns of AMPAR Plasticity in Nucleus Accumbens Medium Spiny Neurons.Gbeta5 recruits R7 RGS proteins to GIRK channels to regulate the timing of neuronal inhibitory signaling.D1 dopamine receptor activation of NFAT-mediated striatal gene expression.Endogenous dopamine and endocannabinoid signaling mediate cocaine-induced reversal of AMPAR synaptic potentiation in the nucleus accumbens shell.Presynaptic contributions of chordin to hippocampal plasticity and spatial learning.Opioid and Psychostimulant Plasticity: Targeting Overlap in Nucleus Accumbens Glutamate Signaling.Hippocampal long-term potentiation is normal in heme oxygenase-2 mutant mice.Prolonged abstinence from cocaine or morphine disrupts separable valuations during decision conflict.Beyond simple tests of value: measuring addiction as a heterogeneous disease of computation-specific valuation processesThe molecular switch hypothesis fails to explain the inconsistent effects of the metabotropic glutamate receptor antagonist MCPG on long-term potentiationOpioid-Mediated Astrocyte-Neuron Signaling in the Nucleus AccumbensSynaptic Depotentiation and mGluR5 Activity in the Nucleus Accumbens Drive Cocaine-Primed Reinstatement of Place Preference
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description
researcher
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wetenschapper
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հետազոտող
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name
Mark J. Thomas
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Mark J. Thomas
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Mark J. Thomas
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Mark J. Thomas
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Mark J. Thomas
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type
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Mark J. Thomas
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Mark J. Thomas
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Mark J. Thomas
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Mark J. Thomas
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Mark J. Thomas
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Mark J. Thomas
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Mark J. Thomas
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Mark J. Thomas
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Mark J. Thomas
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Mark J. Thomas
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P106
P1153
7404755763
P31
P496
0000-0001-5721-5350