about
Muscle-specific Drp1 overexpression impairs skeletal muscle growth via translational attenuation.Mitochondrial complex I deficiency enhances skeletal myogenesis but impairs insulin signaling through SIRT1 inactivation.Deficient nitric oxide signalling impairs skeletal muscle growth and performance: involvement of mitochondrial dysregulation.Low-level laser irradiation modulates cell viability and creatine kinase activity in C2C12 muscle cells during the differentiation process.Mitochondrial reshaping accompanies neural differentiation in the developing spinal cord.AMP-activated protein kinase stimulates Warburg-like glycolysis and activation of satellite cells during muscle regenerationMitophagy is required for mitochondrial biogenesis and myogenic differentiation of C2C12 myoblasts.PGC-1α modulates necrosis, inflammatory response, and fibrotic tissue formation in injured skeletal muscle.FTO is required for myogenesis by positively regulating mTOR-PGC-1α pathway-mediated mitochondria biogenesis.Creatine Prevents the Structural and Functional Damage to Mitochondria in Myogenic, Oxidatively Stressed C2C12 Cells and Restores Their Differentiation Capacity.Down-regulation of the mitochondrial matrix peptidase ClpP in muscle cells causes mitochondrial dysfunction and decreases cell proliferation.Autophagy is required and protects against apoptosis during myoblast differentiation.Drought and host selection influence bacterial community dynamics in the grass root microbiome.Chronic Binge Alcohol-Induced Dysregulation of Mitochondrial-Related Genes in Skeletal Muscle of Simian Immunodeficiency Virus-Infected Rhesus Macaques at End-Stage Disease.Mammalian mitophagy - from in vitro molecules to in vivo models.Methylmercury exposure causes a persistent inhibition of myogenin expression and C2C12 myoblast differentiation.Programmed mitophagy is essential for the glycolytic switch during cell differentiation.Plasticity of the Muscle Stem Cell Microenvironment.Myoblast mitochondrial respiration is decreased in chronic binge alcohol administered simian immunodeficiency virus-infected antiretroviral-treated rhesus macaques.Sphingosine-1-phosphate pretreatment amends hypoxia-induced metabolic dysfunction and impairment of myogenic potential in differentiating C2C12 myoblasts by stimulating viability, calcium homeostasis and energy generation.Analysis of Mitochondrial Network Morphology in Cultured Myoblasts from Patients with Mitochondrial DisordersMitochondrial dysfunction reduces the activity of KIR2.1 K channel in myoblasts impaired oxidative phosphorylation
P2860
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P2860
description
2013 nî lūn-bûn
@nan
2013年の論文
@ja
2013年学术文章
@wuu
2013年学术文章
@zh
2013年学术文章
@zh-cn
2013年学术文章
@zh-hans
2013年学术文章
@zh-my
2013年学术文章
@zh-sg
2013年學術文章
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2013年學術文章
@zh-hant
name
Mitochondria as a potential regulator of myogenesis.
@en
Mitochondria as a potential regulator of myogenesis.
@nl
type
label
Mitochondria as a potential regulator of myogenesis.
@en
Mitochondria as a potential regulator of myogenesis.
@nl
prefLabel
Mitochondria as a potential regulator of myogenesis.
@en
Mitochondria as a potential regulator of myogenesis.
@nl
P2860
P356
P1476
Mitochondria as a potential regulator of myogenesis.
@en
P2093
Akira Wagatsuma
P2860
P304
P356
10.1155/2013/593267
P407
P577
2013-02-03T00:00:00Z