Spinal PKCα inhibition and gene-silencing for pain relief: AMPAR trafficking at the synapses between primary afferents and sensory interneurons.

about

Spinal PKCα inhibition and gene-silencing for pain relief: AMPAR trafficking at the synapses between primary afferents and sensory interneurons.

Item

http://www.wikidata.org/entity/Q55662826

description

2018 nî lūn-bûn

@nan

2018年の論文

@ja

2018年学术文章

@wuu

2018年学术文章

@zh-cn

2018年学术文章

@zh-hans

2018年学术文章

@zh-my

2018年学术文章

@zh-sg

2018年學術文章

@yue

2018年學術文章

@zh

2018年學術文章

@zh-hant

name

Spinal PKCα inhibition and gen ...... ents and sensory interneurons.

@en

Spinal PKCα inhibition and gen ...... ents and sensory interneurons.

@nl

type

Item

label

Spinal PKCα inhibition and gen ...... ents and sensory interneurons.

@en

Spinal PKCα inhibition and gen ...... ents and sensory interneurons.

@nl

prefLabel

Spinal PKCα inhibition and gen ...... ents and sensory interneurons.

@en

Spinal PKCα inhibition and gen ...... ents and sensory interneurons.

@nl

P1476

Spinal PKCα inhibition and gen ...... rents and sensory interneurons

@en

P2093

Andrij Sotnic

http://www.w3.org/2001/XMLSchema#string

Angela Shysh

http://www.w3.org/2001/XMLSchema#string

Nana Voitenko

http://www.w3.org/2001/XMLSchema#string

Viacheslav Viatchenko-Karpinski

http://www.w3.org/2001/XMLSchema#string

Victor Dosenko

http://www.w3.org/2001/XMLSchema#string

P2860

Preserved acute pain and reduced neuropathic pain in mice lacking PKCgamma

Regulation of AMPA receptor extrasynaptic insertion by 4.1N, phosphorylation and palmitoylation.

Synaptic incorporation of AMPA receptors during LTP is controlled by a PKC phosphorylation site on GluR1.

Inflammation alters trafficking of extrasynaptic AMPA receptors in tonically firing lamina II neurons of the rat spinal dorsal horn.

Isozyme-specific effects of protein kinase C in pain modulation.

Phosphorylation of NR2B NMDA subunits by protein kinase C in arcuate nucleus contributes to inflammatory pain in rats

Development of inflammation-induced hyperalgesia and allodynia is associated with the upregulation of extrasynaptic AMPA receptors in tonically firing lamina II dorsal horn neurons.

Protein kinase C gamma-mediated phosphorylation of GluA1 in the postsynaptic density of spinal dorsal horn neurons accompanies neuropathic pain, and dephosphorylation by calcineurin is associated with prolonged analgesia.

PKCα is required for inflammation-induced trafficking of extrasynaptic AMPA receptors in tonically firing lamina II dorsal horn neurons during the maintenance of persistent inflammatory pain

Inhibition of Spinal Ca(2+)-Permeable AMPA Receptors with Dicationic Compounds Alleviates Persistent Inflammatory Pain without Adverse Effects.

P304

10285

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

P356

10.1038/S41598-018-28512-9

http://www.w3.org/2001/XMLSchema#string

P407

English

P433

http://www.w3.org/2001/XMLSchema#string

P478

http://www.w3.org/2001/XMLSchema#string

P50

Olga Kopach

Volodymyr Krotov

P577

2018-07-06T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P698

29980697

http://www.w3.org/2001/XMLSchema#string

P932

6035211

http://www.w3.org/2001/XMLSchema#string