about
Novel markers of early ovarian pre-granulosa cells are expressed in an Sry-like patternNHERF2/SIP-1 interacts with mouse SRY via a different mechanism than human SRY.DNA sequence analysis of Sry alleles (subgenus Mus) implicates misregulation as the cause of C57BL/6J-Y(POS) sex reversal and defines the SRY functional unitSex reversal in C57BL/6J XY mice caused by increased expression of ovarian genes and insufficient activation of the testis determining pathway.C57BL/6J-T-associated sex reversal in mice is caused by reduced expression of a Mus domesticus Sry allele.Sry expression level and protein isoform differences play a role in abnormal testis development in C57BL/6J mice carrying certain Sry allelesDevelopmental expression of Smoc1 and Smoc2 suggests potential roles in fetal gonad and reproductive tract differentiation.Deletion of Cavin/PTRF causes global loss of caveolae, dyslipidemia, and glucose intolerance.Correct dosage of Fog2 and Gata4 transcription factors is critical for fetal testis development in mice.The developmentally-regulated Smoc2 gene is repressed by Aryl-hydrocarbon receptor (Ahr) signaling.Evidence that Sry is expressed in pre-Sertoli cells and Sertoli and granulosa cells have a common precursor.Migration of mesonephric cells into the mammalian gonad depends on Sry.
P50
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P50
description
researcher
@en
wetenschapper
@nl
հետազոտող
@hy
name
Kenneth H Albrecht
@ast
Kenneth H Albrecht
@en
Kenneth H Albrecht
@es
Kenneth H Albrecht
@nl
Kenneth H Albrecht
@sl
type
label
Kenneth H Albrecht
@ast
Kenneth H Albrecht
@en
Kenneth H Albrecht
@es
Kenneth H Albrecht
@nl
Kenneth H Albrecht
@sl
prefLabel
Kenneth H Albrecht
@ast
Kenneth H Albrecht
@en
Kenneth H Albrecht
@es
Kenneth H Albrecht
@nl
Kenneth H Albrecht
@sl
P106
P31
P496
0000-0002-9769-3362