about
Salvianolic acid B inhibits hydrogen peroxide-induced endothelial cell apoptosis through regulating PI3K/Akt signalingEffects of Huanglian-Jie-Du-Tang and its modified formula on the modulation of amyloid-β precursor protein processing in Alzheimer's disease modelsSalvianolic acid B inhibits Abeta fibril formation and disaggregates preformed fibrils and protects against Abeta-induced cytotoxicty.Efficacy and safety of acupuncture for idiopathic Parkinson's disease: a systematic review.Treatment of idiopathic Parkinson's disease with traditional chinese herbal medicine: a randomized placebo-controlled pilot clinical study.Tianma Gouteng Yin, a Traditional Chinese Medicine decoction, exerts neuroprotective effects in animal and cellular models of Parkinson's disease.Corynoxine, a natural autophagy enhancer, promotes the clearance of alpha-synuclein via Akt/mTOR pathway.Berberine ameliorates β-amyloid pathology, gliosis, and cognitive impairment in an Alzheimer's disease transgenic mouse model.HMGB1 is involved in autophagy inhibition caused by SNCA/α-synuclein overexpression: a process modulated by the natural autophagy inducer corynoxine B.Isorhynchophylline, a natural alkaloid, promotes the degradation of alpha-synuclein in neuronal cells via inducing autophagy.In vitro screening on amyloid precursor protein modulation of plants used in Ayurvedic and traditional Chinese medicine for memory improvement.A modified formulation of Huanglian-Jie-Du-Tang reduces memory impairments and β-amyloid plaques in a triple transgenic mouse model of Alzheimer's diseaseDecrease in the Generation of Amyloid-β Due to Salvianolic Acid B by Modulating BACE1 Activity.Corynoxine isomers decrease levels of amyloid-β peptide and amyloid-β precursor protein by promoting autophagy and lysosome biogenesisThe effect of salvianolic acid B combined with laminar shear stress on TNF-alpha-stimulated adhesion molecule expression in human aortic endothelial cellsChronic oral administration of adipoRon reverses cognitive impairments and ameliorates neuropathology in an Alzheimer's disease mouse modelNRBF2 is a RAB7 effector required for autophagosome maturation and mediates the association of APP-CTFs with active form of RAB7 for degradation
P50
Q21144404-CEC5C7E9-9569-468C-83D5-ED987933CD48Q28541573-0F40662D-EE88-4A02-A987-D72338745B6CQ33304138-539C5759-5F04-496D-97A2-BA0F0EB353AEQ33358282-E9673E58-F22B-4162-BBA8-99EF2253EDD3Q35108175-158BF162-E5F5-4682-9578-14E20C636854Q38818810-F2374802-85D1-49D8-B3B6-5C2C632A6398Q39024437-83CA1DAA-FBAC-40A3-B869-17EBF109B12FQ39372237-2EC0F76D-7D28-4962-B75A-5ADB40818789Q39394670-DC97F0D9-67C9-4AEC-88F1-FF0E2E9CB807Q39437985-F542284A-9D42-47D8-95A6-28561B493289Q39473403-52407810-2B15-4D0A-AF38-437FF425559FQ41109313-BF424053-CFA4-4885-9869-416146D932E1Q51055104-69D2ACA0-A942-4D14-AD55-E7EE22F8ED26Q59299085-E7D54E84-9EE8-41C5-970C-61E292A764F2Q84461863-BCFE1C66-5053-487C-8AA6-1D8765EEF87BQ90053568-8BC4776B-7373-42E4-B3EE-98829F7ED04EQ96435278-BA11C4D2-7020-423A-BECF-AB2F4F0C7D5B
P50
description
onderzoeker
@nl
researcher
@en
հետազոտող
@hy
name
Siva Sundara Kumar Durairajan
@ast
Siva Sundara Kumar Durairajan
@en
Siva Sundara Kumar Durairajan
@es
Siva Sundara Kumar Durairajan
@nl
Siva Sundara Kumar Durairajan
@sl
type
label
Siva Sundara Kumar Durairajan
@ast
Siva Sundara Kumar Durairajan
@en
Siva Sundara Kumar Durairajan
@es
Siva Sundara Kumar Durairajan
@nl
Siva Sundara Kumar Durairajan
@sl
prefLabel
Siva Sundara Kumar Durairajan
@ast
Siva Sundara Kumar Durairajan
@en
Siva Sundara Kumar Durairajan
@es
Siva Sundara Kumar Durairajan
@nl
Siva Sundara Kumar Durairajan
@sl
P106
P31
P4012
P496
0000-0001-7376-7163