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Free Radical Mechanisms of Aging Processes Under Physiological Conditions

Free Radical Mechanisms of Aging Processes Under Physiological Conditions

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Impaired Autophagy and Defective Mitochondrial Function: Converging Paths on the Road to Motor Neuron Degeneration Iron behaving badly: inappropriate iron chelation as a major contributor to the aetiology of vascular and other progressive inflammatory and degenerative diseases Catalase takes part in rat liver mitochondria oxidative stress defense Altered mitochondrial function in type 2 granular corneal dystrophy Antioxidant-induced stress.The anti-aging properties of a human placental hydrolysate combined with dieckol isolated from Ecklonia cava.Signaling and Damaging Functions of Free Radicals in Aging-Free Radical Theory, Hormesis, and TOR.Biomedical studies on lipid peroxidation and erythrocyte fragility during the process of aging.Reactive oxygen species and age-related genes p66shc, Sirtuin, FOX03 and Klotho in senescence.Age related changes in lipid peroxidation and antioxidants in elderly people.p66Shc Inactivation Modifies RNS Production, Regulates Sirt3 Activity, and Improves Mitochondrial Homeostasis, Delaying the Aging Process in Mouse Brain.

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P2860

Free Radical Mechanisms of Aging Processes Under Physiological Conditions

http://www.wikidata.org/entity/Q56384848

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wetenschappelijk artikel

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наукова стаття, опублікована в липні 2005

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name

Free Radical Mechanisms of Aging Processes Under Physiological Conditions

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Free Radical Mechanisms of Aging Processes Under Physiological Conditions

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type

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Free Radical Mechanisms of Aging Processes Under Physiological Conditions

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Free Radical Mechanisms of Aging Processes Under Physiological Conditions

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Free Radical Mechanisms of Aging Processes Under Physiological Conditions

@en

Free Radical Mechanisms of Aging Processes Under Physiological Conditions

@nl

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Free Radical Mechanisms of Aging Processes Under Physiological Conditions

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Igor B Afanas’ev

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P2860

Cytochrome c release from mitochondria in the aging heart: a possible mechanism for apoptosis with age

Superoxide excess in hypertension and aging: a common cause of endothelial dysfunction.

Age-related reduction of NO availability and oxidative stress in humans.

High rates of extracellular superoxide generation by cultured human fibroblasts: involvement of a lipid-metabolizing enzyme.

Alterations of nitric oxide synthase expression with aging and hypertension in rats.

Role of oxidative stress in age-related reduction of NO-cGMP-mediated vascular relaxation in SHR.

Selected Contribution: Aging impairs nitric oxide and prostacyclin mediation of endothelium-dependent dilation in soleus feed arteries.

NAD(P)H oxidase-generated superoxide anion accounts for reduced control of myocardial O2 consumption by NO in old Fischer 344 rats.

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s10522-005-2626-z

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283-290

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scholarly article

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10.1007/S10522-005-2626-Z

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4

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2005-07-01T00:00:00Z

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7435403

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16333762

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