about
Persistence of pulmonary tertiary lymphoid tissues and anti-nuclear antibodies following cessation of cigarette smoke exposureTreating viral exacerbations of chronic obstructive pulmonary disease: insights from a mouse model of cigarette smoke and H1N1 influenza infectionAlterations in skeletal muscle cell homeostasis in a mouse model of cigarette smoke exposure.Induction of the interleukin 6/ signal transducer and activator of transcription pathway in the lungs of mice sub-chronically exposed to mainstream tobacco smoke.Impact of inflammation, emphysema, and smoking cessation on V/Q in mouse models of lung obstruction.The role of the CCR1 receptor in the inflammatory response to tobacco smoke in a mouse modelContinuous and discontinuous cigarette smoke exposure differentially affects protective Th1 immunity against pulmonary tuberculosis.Influenza promotes collagen deposition via αvβ6 integrin-mediated transforming growth factor β activation.On the generation of allergic airway diseases: from GM-CSF to Kyoto.A GM-CSF/IL-33 pathway facilitates allergic airway responses to sub-threshold house dust mite exposureImpact of cigarette smoke on the human and mouse lungs: a gene-expression comparison study.Estradiol Enhances CD4+ T-Cell Anti-Viral Immunity by Priming Vaginal DCs to Induce Th17 Responses via an IL-1-Dependent PathwayCigarette smoke exposure triggers the autophagic cascade via activation of the AMPK pathway in mice.Cloning of human anti-IgE autoantibodies and their role in the regulation of IgE synthesis.Animal models of chronic obstructive pulmonary disease exacerbations.Induction of pulmonary antibodies against oxidized lipids in mice exposed to cigarette smoke.Cigarette smoke-induced inflammation and respiratory host defense: Insights from animal models.Targeting Interleukin-17 signalling in cigarette smoke-induced lung disease: Mechanistic concepts and therapeutic opportunities.Cigarette smoke and platelet-activating factor receptor dependent adhesion of Streptococcus pneumoniae to lower airway cells.Detection of lung dysfunction using ventilation and perfusion SPECT in a mouse model of chronic cigarette smoke exposure.Acute, but not resolved, influenza A infection enhances susceptibility to house dust mite-induced allergic disease.Generation of experimental allergic airways inflammation in the absence of draining lymph nodes.Cigarette smoke suppresses type I interferon-mediated antiviral immunity in lung fibroblast and epithelial cells.Streptococcus pneumoniae colonization is required to alter the nasal microbiota in cigarette smoke-exposed mice.Cigarette smoke impairs NK cell-dependent tumor immune surveillance.Biological activities of anti-IgE antibodies.IL-17A and the Promotion of Neutrophilia in Acute Exacerbation of Chronic Obstructive Pulmonary Disease.Neuropeptides accentuate interleukin-4 induced human immunoglobuline E synthesis in vitro.Hydroxychloroquine attenuates cigarette smoke induced autophagic signaling in the mouse ovary.A novel role for IL-17 in enhancing type 1 helper T cell immunity in the female genital tract following mucosal HSV-2 vaccination.Correction: Estradiol Enhances CD4+ T-Cell Anti-Viral Immunity by Priming Vaginal DCs to Induce Th17 Responses via an IL-1-Dependent Pathway.Cigarette smoke exposure elicits increased autophagy and dysregulation of mitochondrial dynamics in murine granulosa cells.Cigarette smoke attenuation of poly I:C-induced innate antiviral responses in human PBMC is mainly due to inhibition of IFN-beta production.Impairment of human NK cell cytotoxic activity and cytokine release by cigarette smoke.Identification of Drug Candidates to Suppress Cigarette Smoke-induced Inflammation via cMap Analyses.Inhalation tolerance is induced selectively in thoracic lymph nodes but executed pervasively at distant mucosal and nonmucosal tissues.B7RP-1 is not required for the generation of Th2 responses in a model of allergic airway inflammation but is essential for the induction of inhalation tolerance.The lung cytokine microenvironment influences molecular events in the lymph nodes during Th1 and Th2 respiratory mucosal sensitization to antigen in vivo.Concomitant airway expression of granulocyte-macrophage colony-stimulating factor and decorin, a natural inhibitor of transforming growth factor-beta, breaks established inhalation tolerance.Disruption of pulmonary lipid homeostasis drives cigarette smoke-induced lung inflammation in mice.
P50
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P50
description
researcher
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wetenschapper
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հետազոտող
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name
Martin R Stampfli
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Martin R Stampfli
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Martin R Stampfli
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Martin R Stampfli
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type
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Martin R Stampfli
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Martin R Stampfli
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Martin R Stampfli
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Martin R Stampfli
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Martin R Stampfli
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Martin R Stampfli
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Martin R Stampfli
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Martin R Stampfli
@nl
P106
P21
P31
P496
0000-0003-4918-5889