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Functional analysis of HBV genomes from patients with fulminant hepatitis

Functional analysis of HBV genomes from patients with fulminant hepatitis

http://www.wikidata.org/entity/Q56439587

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Genomic Diversity of Hepatitis B Virus Infection Associated With Fulminant Hepatitis B Development Molecular epidemiological study on pre-X region of hepatitis B virus and identification of hepatocyte proteins interacting with whole-X protein by yeast two-hybrid The core promoter of hepatitis B virus.Screening and identification of interacting proteins with hepatitis B virus core protein in leukocytes and cloning of new gene C1.Control or persistence of hepatitis B virus: the critical role of initial host-virus interactions.Occult hepatitis B virus infection and its clinical implications.Distribution of hepatitis B viral genotypes and mutations in the core promoter and precore regions in acute forms of liver disease in patients from Chiba, Japan Strong association between genotype F and hepatitis B virus (HBV) e antigen-negative variants among HBV-infected argentinean blood donors.Hepatocellular carcinoma: the point of view of the hepatitis B virus.Renal transplantation from hepatitis B surface antigen (HBsAg)-positive donors to HBsAg-negative recipients: a case of post-transplant fulminant hepatitis associated with an extensively mutated hepatitis B virus strain and review of the current lite Core promoter: a critical region where the hepatitis B virus makes decisions.Molecular epidemiology of an outbreak of fulminant hepatitis B.Hepatitis B reactivation characterized by HBsAg negativity and anti-HbsAg antibodies persistence in haematopoietic stem cell transplanted patient after lamivudine withdrawal.Distinct quasispecies characteristics and positive selection within precore/core gene in hepatitis B virus HBV associated acute-on-chronic liver failure.Chronic hepatitis B carriers with acute on chronic liver failure show increased HBV surface gene mutations, including immune escape variants.Molecular, immunological and clinical properties of mutated hepatitis B viruses.Infectious source factors affecting the severity of sexually transmitted acute hepatitis due to hepatitis B virus genotype C.Detection of mutations in hepatitis B virus enhancer 2/core promoter and X protein regions in patients with fatal hepatitis B virus infection.Mutations and deletions in core promoter and precore stop codon in relation to viral replication and liver damage in Singaporean hepatitis B virus carriers.Pathogenesis of hepatitis B virus infection.Molecular Variants of the Precore, Core, and Core Promoter Regions of Hepatitis B Virus, and Their Clinical Significance

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Functional analysis of HBV genomes from patients with fulminant hepatitis

http://www.wikidata.org/entity/Q56439587

description

article

@en

im November 1998 veröffentlichter wissenschaftlicher Artikel

@de

wetenschappelijk artikel

@nl

наукова стаття, опублікована в листопаді 1998

@uk

ലേഖനം

@ml

name

Functional analysis of HBV genomes from patients with fulminant hepatitis

@en

Functional analysis of HBV genomes from patients with fulminant hepatitis

@nl

type

label

Functional analysis of HBV genomes from patients with fulminant hepatitis

@en

Functional analysis of HBV genomes from patients with fulminant hepatitis

@nl

prefLabel

Functional analysis of HBV genomes from patients with fulminant hepatitis

@en

Functional analysis of HBV genomes from patients with fulminant hepatitis

@nl

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Functional analysis of HBV genomes from patients with fulminant hepatitis

@en

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Hans Will

http://www.w3.org/2001/XMLSchema#string

Heiner Greten

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Lutz Fischer

http://www.w3.org/2001/XMLSchema#string

Martina Sterneck

http://www.w3.org/2001/XMLSchema#string

Tatjana Kalinina

http://www.w3.org/2001/XMLSchema#string

Teresa Santantonio

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P2860

Multiple liver-specific factors bind to the hepatitis B virus core/pregenomic promoter: trans-activation and repression by CCAAT/enhancer binding protein.

Mutations in core nucleotide sequence of hepatitis B virus correlate with fulminant and severe hepatitis.

Nucleotide sequence of the hepatitis B virus genome (subtype ayw) cloned in E. coli.

Hepatitis B virus precore mutation and fulminant hepatitis in the United States. A polymerase chain reaction-based assay for the detection of specific mutation.

Mechanisms of class I restricted immunopathology. A transgenic mouse model of fulminant hepatitis

A liver-specific nuclear factor interacts with the promoter region of the large surface protein gene of human hepatitis B virus

Two core promotor mutations identified in a hepatitis B virus strain associated with fulminant hepatitis result in enhanced viral replication.

Reduced precore transcription and enhanced core-pregenome transcription of hepatitis B virus DNA after replacement of the precore-core promoter with sequences associated with e antigen-seronegative persistent infections.

Fulminant hepatitis B: induction by hepatitis B virus mutants defective in the precore region and incapable of encoding e antigen.

Secretion and antigenicity of hepatitis B virus small envelope proteins lacking cysteines in the major antigenic region.

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1390-1397

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scholarly article

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10.1002/HEP.510280530

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5

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28

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Stephan Günther

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1998-11-01T00:00:00Z

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227788488

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9794926

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