about
Autonomic, locomotor and cardiac abnormalities in a mouse model of muscular dystrophy: targeting the renin-angiotensin systemThe ion channel ASIC2 is required for baroreceptor and autonomic control of the circulation.Exercise prevents development of autonomic dysregulation and hyperalgesia in a mouse model of chronic muscle pain.Regulator of G protein signaling 2 deficiency causes endothelial dysfunction and impaired endothelium-derived hyperpolarizing factor-mediated relaxation by dysregulating Gi/o signalingChronic oral administration of Ang-(1-7) improves skeletal muscle, autonomic and locomotor phenotypes in muscular dystrophy.Methods of assessing vagus nerve activity and reflexes.Angiotensin-dependent autonomic dysregulation precedes dilated cardiomyopathy in a mouse model of muscular dystrophy.The link between stress disorders and autonomic dysfunction in muscular dystrophyReceptor activity-modifying protein 1 increases baroreflex sensitivity and attenuates Angiotensin-induced hypertension.Autonomic regulation in muscular dystrophy.Effect of hypothermia on baroreflex control of heart rate and renal sympathetic nerve activity in anaesthetized rats.Cold-impaired cardiac performance in rats is only partially overcome by cold acclimation.Increased receptor activity-modifying protein 1 in the nervous system is sufficient to protect against autonomic dysregulation and hypertension.
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description
researcher
@en
wetenschapper
@nl
հետազոտող
@hy
name
Rasna Sabharwal
@ast
Rasna Sabharwal
@en
Rasna Sabharwal
@es
Rasna Sabharwal
@nl
type
label
Rasna Sabharwal
@ast
Rasna Sabharwal
@en
Rasna Sabharwal
@es
Rasna Sabharwal
@nl
prefLabel
Rasna Sabharwal
@ast
Rasna Sabharwal
@en
Rasna Sabharwal
@es
Rasna Sabharwal
@nl
P106
P1153
8096675400
P31
P496
0000-0001-7256-5359