about
Interferon-gamma release assay (modified QuantiFERON) as a potential marker of infection for Leishmania donovani, a proof of concept studyCombined Immune Therapy for the Treatment of Visceral LeishmaniasisEvaluation of ex vivo human immune response against candidate antigens for a visceral leishmaniasis vaccineMeasurement of recent exposure to Phlebotomus argentipes, the vector of Indian visceral Leishmaniasis, by using human antibody responses to sand fly salivaImmune regulation during chronic visceral leishmaniasis.Leishmania specific CD4 T cells release IFNγ that limits parasite replication in patients with visceral leishmaniasis.IgG1 as a potential biomarker of post-chemotherapeutic relapse in visceral leishmaniasis, and adaptation to a rapid diagnostic testLonglasting insecticidal nets for prevention of Leishmania donovani infection in India and Nepal: paired cluster randomised trial.Enhanced expression of Toll-like receptors 2 and 4, but not 9, in spleen tissue from patients with visceral leishmaniasis.Immunobiology of visceral leishmaniasis.Persistence of Leishmania donovani antibodies in past visceral leishmaniasis cases in IndiaIL-27 and IL-21 are associated with T cell IL-10 responses in human visceral leishmaniasis.Significantly lower anti-Leishmania IgG responses in Sudanese versus Indian visceral leishmaniasis.IL-10 neutralization promotes parasite clearance in splenic aspirate cells from patients with visceral leishmaniasis.Blimp-1-Dependent IL-10 Production by Tr1 Cells Regulates TNF-Mediated Tissue Pathology.Reassessment of immune correlates in human visceral leishmaniasis as defined by cytokine release in whole bloodType I Interferons Regulate Immune Responses in Humans with Blood-Stage Plasmodium falciparum Infection.CD8 T cell exhaustion in human visceral leishmaniasis.Evaluation of rk39 immunochromatographic test with urine for diagnosis of visceral leishmaniasis.Tumor necrosis factor alpha neutralization has no direct effect on parasite burden, but causes impaired IFN-γ production by spleen cells from human visceral leishmaniasis patients.IL-17A-Producing γδ T Cells Suppress Early Control of Parasite Growth by Monocytes in the Liver.Mast cells fuel the fire of malaria immunopathology.Rapid loss of group 1 innate lymphoid cells during blood stage Plasmodium infection‡.Distinct Roles for CD4 Foxp3 Regulatory T Cells and IL-10-Mediated Immunoregulatory Mechanisms during Experimental Visceral Leishmaniasis Caused byThe Role of BACH2 in T Cells in Experimental Malaria Caused by ASThe Role of IL-10 in Malaria: A Double Edged SwordPeripheral Blood Monocytes With an Antiinflammatory Phenotype Display Limited Phagocytosis and Oxidative Burst in Patients With Visceral LeishmaniasisThe regulation of CD4+ T cells during malariaInterleukin 2 is an Upstream Regulator of CD4+ T Cells From Visceral Leishmaniasis Patients With Therapeutic Potential
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P50
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researcher ORCID: 0000-0003-2338-1494
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Rajiv Kumar
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0000-0003-2338-1494